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部分肌腱撕裂后的触发机制。

Triggering after partial tendon laceration.

作者信息

al-Qattan M M, Posnick J C, Lin K Y

机构信息

Division of Plastic Surgery, University of Toronto, Ontario, Canada.

出版信息

J Hand Surg Br. 1993 Apr;18(2):241-6. doi: 10.1016/0266-7681(93)90119-z.

Abstract

Triggering and tendon flap formation were studied after a transverse laceration of 50% of the width of the flexor digitorum profundus tendons of the hind limb of 14 adult sheep at various intervals after injury. The tendon laceration was not repaired and there was no post-operative immobilization. Triggering was not caused by bulbous scar formation but by the bunching of the tendon fibres proximal or distal to the laceration site. This bunched part of the tendon appeared to become incorporated into the healing process, with gradual spontaneous resolution of triggering. Failure of incorporation of this bunched part resulted in the formation of a flap in two tendons.

摘要

在14只成年绵羊后肢的指深屈肌腱宽度50%处进行横向撕裂伤,并在损伤后的不同时间间隔对触发和肌腱瓣形成进行了研究。肌腱撕裂未进行修复,且术后未进行固定。触发不是由球茎状瘢痕形成引起的,而是由撕裂部位近端或远端的肌腱纤维束状聚集引起的。肌腱的这一束状部分似乎融入了愈合过程,触发逐渐自发消退。这一束状部分未融入导致两根肌腱形成了一个瓣。

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