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不稳定型心绞痛中凝血系统的间歇性激活不会引发急性期反应。

Episodic activation of the coagulation system in unstable angina does not elicit an acute phase reaction.

作者信息

Biasucci L M, Liuzzo G, Caligiuri G, van de Greef W, Quaranta G, Monaco C, Rebuzzi A G, Kluft C, Maseri A

机构信息

Institute of Cardiology, Catholic University of the Sacred Heart, Rome, Italy.

出版信息

Am J Cardiol. 1996 Jan 1;77(1):85-7. doi: 10.1016/s0002-9149(97)89141-2.

DOI:10.1016/s0002-9149(97)89141-2
PMID:8540465
Abstract

The results of our study suggest that the acute phase response may be partly related to a yet unknown primary inflammatory component in unstable angina. Further studies are needed to elucidate the actual role of inflammation in unstable angina and its relation to activation of the coagulation system.

摘要

我们的研究结果表明,急性期反应可能部分与不稳定型心绞痛中一种尚不清楚的原发性炎症成分有关。需要进一步研究以阐明炎症在不稳定型心绞痛中的实际作用及其与凝血系统激活的关系。

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1
Episodic activation of the coagulation system in unstable angina does not elicit an acute phase reaction.不稳定型心绞痛中凝血系统的间歇性激活不会引发急性期反应。
Am J Cardiol. 1996 Jan 1;77(1):85-7. doi: 10.1016/s0002-9149(97)89141-2.
2
Temporal relation between ischemic episodes and activation of the coagulation system in unstable angina.不稳定型心绞痛中缺血发作与凝血系统激活之间的时间关系。
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Prothrombin fragment 1 + 2 and thrombin-antithrombin III complex as markers of activation of blood coagulation in inflammatory bowel diseases.凝血酶原片段1+2和凝血酶-抗凝血酶III复合物作为炎症性肠病中血液凝固激活的标志物。
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Measurement of markers of activated coagulation in antithrombin III deficient subjects.抗凝血酶III缺乏症患者中活化凝血标志物的测定。
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Short-time stability of markers of coagulation and fibrinolysis in frozen plasma.冷冻血浆中凝血和纤溶标志物的短期稳定性
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Modified antithrombin III levels during normal pregnancy and relationship with prothrombin fragment F1 + 2 and thrombin-antithrombin complexes.正常妊娠期间抗凝血酶III水平的变化及其与凝血酶原片段F1 + 2和凝血酶-抗凝血酶复合物的关系。
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Inflammatory status as a main determinant of outcome in patients with unstable angina, independent of coagulation activation and endothelial cell function.炎症状态是不稳定型心绞痛患者预后的主要决定因素,独立于凝血激活和内皮细胞功能。
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Human endothelial cells suppress prothrombin activation in nonanticoagulated whole blood in vitro.人内皮细胞在体外可抑制非抗凝全血中的凝血酶原激活。
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Factor XIa and Thrombin Generation Are Elevated in Patients with Acute Coronary Syndrome and Predict Recurrent Cardiovascular Events.急性冠脉综合征患者的因子XIa和凝血酶生成升高,并可预测心血管事件复发。
PLoS One. 2016 Jul 15;11(7):e0158355. doi: 10.1371/journal.pone.0158355. eCollection 2016.
2
Relationship between minor myocardial damage and inflammatory acute-phase reaction in acute coronary syndromes.急性冠状动脉综合征中轻微心肌损伤与炎症急性期反应的关系
J Thromb Thrombolysis. 2003 Feb;15(1):33-9. doi: 10.1023/a:1026140317777.
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Inflammation in ischaemic heart disease.
缺血性心脏病中的炎症
BMJ. 1996 Apr 27;312(7038):1049-50. doi: 10.1136/bmj.312.7038.1049.