细胞间黏附分子-1（ICAM-1）在大鼠反向被动阿瑟斯反应诱导的胸膜炎中的作用。 - Suppr

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超能文献

The role of ICAM-1 in the reverse passive Arthus reaction induced pleurisy in the rat.

作者信息

Fielding L E, Buckley S K, Perez M, Grimes D, Bard F, Gordan G, Horner H, Yednock T

机构信息

Wyeth-Ayerst Research, Princeton, NJ 08543-8000, USA.

出版信息

Inflamm Res. 1995 Aug;44 Suppl 2:S213-4. doi: 10.1007/BF01778339.

DOI:10.1007/BF01778339

PMID:8548405

Abstract

摘要

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本文引用的文献

Adhesion molecules and inflammatory injury.黏附分子与炎症损伤

FASEB J. 1994 May;8(8):504-12.

Differential effects of anti-inflammatory drugs on fluid accumulation and cellular infiltration in reverse passive arthus pleurisy and carrageenan pleurisy in rats.抗炎药物对大鼠反向被动阿瑟斯胸膜炎和角叉菜胶胸膜炎中液体蓄积及细胞浸润的不同作用。

Pharmacology. 1987;34(6):309-25. doi: 10.1159/000138285.

Intercellular adhesion molecule-1 (ICAM-1) in the pathogenesis of asthma.细胞间黏附分子-1（ICAM-1）在哮喘发病机制中的作用

Science. 1990 Jan 26;247(4941):456-9. doi: 10.1126/science.1967851.

A model for the quantitative study of Arthus (immunologic) hypersensitivity in rats.

Agents Actions. 1975 Oct;5(4):374-7. doi: 10.1007/BF02205246.

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