A组链球菌表达的纤溶酶受体蛋白的突变分析。 - Suppr

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超能文献

Mutational analysis of a plasmin receptor protein expressed by group A streptococci.

作者信息

Winram S B, Richardson L C, Lottenberg R

机构信息

Department of Medicine, University of Florida College of Medicine, Gainesville, USA.

出版信息

Dev Biol Stand. 1995;85:199-202.

PMID:8586176

Abstract

摘要

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Mutational analysis of a plasmin receptor protein expressed by group A streptococci.A组链球菌表达的纤溶酶受体蛋白的突变分析。

Dev Biol Stand. 1995;85:199-202.

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Site-directed mutagenesis of streptococcal plasmin receptor protein (Plr) identifies the C-terminal Lys334 as essential for plasmin binding, but mutation of the plr gene does not reduce plasmin binding to group A streptococci.对链球菌纤溶酶受体蛋白（Plr）进行定点诱变，确定其C末端的赖氨酸334对纤溶酶结合至关重要，但plr基因突变并不会降低纤溶酶与A组链球菌的结合。

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Characterization of group B streptococcal glyceraldehyde-3-phosphate dehydrogenase: surface localization, enzymatic activity, and protein-protein interactions.B族链球菌3-磷酸甘油醛脱氢酶的特性：表面定位、酶活性及蛋白质-蛋白质相互作用

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A novel plasminogen/plasmin binding protein on the surface of group A streptococci.A群链球菌表面一种新型的纤溶酶原/纤溶酶结合蛋白。

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Iron starvation causes release from the group A streptococcus of the ADP-ribosylating protein called plasmin receptor or surface glyceraldehyde-3-phosphate-dehydrogenase.铁饥饿会导致A组链球菌释放一种名为纤溶酶受体或表面甘油醛-3-磷酸脱氢酶的ADP核糖基化蛋白。

Infect Immun. 1996 Jun;64(6):1956-60. doi: 10.1128/iai.64.6.1956-1960.1996.

The potential role for nephritis-associated plasmin receptor in acute poststreptococcal glomerulonephritis.肾炎相关纤溶酶受体在急性链球菌感染后肾小球肾炎中的潜在作用。

Methods. 2000 Jun;21(2):185-97. doi: 10.1006/meth.2000.0990.

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Role of the C-terminal lysine residues of streptococcal surface enolase in Glu- and Lys-plasminogen-binding activities of group A streptococci.A群链球菌表面烯醇化酶C末端赖氨酸残基在Glu-和Lys-纤溶酶原结合活性中的作用

Infect Immun. 2004 Jan;72(1):94-105. doi: 10.1128/IAI.72.1.94-105.2004.

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