Suwa K
Department of Anaesthesia, Faculty of Medicine, University of Tokyo, Japan.
Acta Anaesthesiol Scand Suppl. 1995;107:215-8. doi: 10.1111/j.1399-6576.1995.tb04361.x.
In order to analyse the effects of various physiological parameters including blood gas data on the cerebral oxygenation, we developed a model to enable us to estimate PtO2 (tissue PO2) from these physiological parameters. This model was applied successfully to a set of blood gas data of a patient who had brain damage after cardiac surgery. Contrary to the original impression, we concluded that a very low PaCO2 of 13 mmHg towards the end of CPB was the most likely cause of his damage.
为了分析包括血气数据在内的各种生理参数对脑氧合的影响，我们开发了一个模型，以便能够根据这些生理参数估算组织氧分压（PtO2）。该模型已成功应用于一名心脏手术后脑部受损患者的一组血气数据。与最初的印象相反，我们得出结论，体外循环末期非常低的动脉血二氧化碳分压（PaCO2）为13 mmHg最有可能是其脑部损伤的原因。
