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人类结核病中缺乏显著的Th2细胞因子反应。

Absence of a prominent Th2 cytokine response in human tuberculosis.

作者信息

Lin Y, Zhang M, Hofman F M, Gong J, Barnes P F

机构信息

Division of Infectious Diseases, Department of Medicine, Los Angeles, California, USA.

出版信息

Infect Immun. 1996 Apr;64(4):1351-6. doi: 10.1128/iai.64.4.1351-1356.1996.

Abstract

Depressed Th1 responses are a prominent feature of human tuberculosis, but an enhanced Th2 response has not been detected in peripheral blood T cells stimulated in vitro with Mycobacterium tuberculosis. In disease due to Mycobacterium leprae, Th2 cells predominate in tissue lesions of patients with extensive disease but are absent from peripheral blood. To determine if Th2 cells are present in tissue lesions of tuberculosis patients, we evaluated patterns of cytokine expression in lymph nodes from tuberculosis patients with or without human immunodeficiency virus infection and in controls without tuberculosis. Gamma interferon and interleukin-10 (IL-10) mRNA expression in tuberculosis patients with or without human immunodeficiency virus infection was high, whereas IL-4 expression in the same patients was low. Immunolabeling studies showed that macrophage production of IL-12 was increased in lymph nodes from tuberculosis patients, that gamma interferon was produced by T cells, and that IL-10 was produced by macrophages rather than Th2 cells. These results indicate that Th2 responses are not enhanced either systemically or at the site of disease in human tuberculosis.

摘要

Th1反应受抑制是人类结核病的一个显著特征,但在用结核分枝杆菌体外刺激的外周血T细胞中未检测到Th2反应增强。在麻风分枝杆菌所致疾病中,Th2细胞在广泛病变患者的组织损伤中占主导,但在外周血中不存在。为了确定Th2细胞是否存在于结核病患者的组织损伤中,我们评估了有或无人免疫缺陷病毒感染的结核病患者以及无结核病的对照者淋巴结中的细胞因子表达模式。有或无人免疫缺陷病毒感染的结核病患者中γ干扰素和白细胞介素-10(IL-10)的mRNA表达较高,而同一患者中IL-4的表达较低。免疫标记研究表明,结核病患者淋巴结中巨噬细胞产生的IL-12增加,γ干扰素由T细胞产生,IL-10由巨噬细胞而非Th2细胞产生。这些结果表明,在人类结核病中,Th2反应在全身或疾病部位均未增强。

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