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Electron microscopy of Lewy bodies in the amygdala-parahippocampal region. Comparison with inclusion bodies in the MPTP-treated squirrel monkey.

作者信息

Forno L S, DeLanney L E, Irwin I, Langston J W

机构信息

VA Medical Center, Palo Alto, California 94304, USA.

出版信息

Adv Neurol. 1996;69:217-28.

PMID:8615131
Abstract
摘要

相似文献

1
Electron microscopy of Lewy bodies in the amygdala-parahippocampal region. Comparison with inclusion bodies in the MPTP-treated squirrel monkey.杏仁核-海马旁回区域路易小体的电子显微镜检查。与MPTP处理的松鼠猴中的包涵体比较。
Adv Neurol. 1996;69:217-28.
2
Similarities and differences between MPTP-induced parkinsonsim and Parkinson's disease. Neuropathologic considerations.MPTP 诱导的帕金森综合征与帕金森病之间的异同。神经病理学考量。
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Ultrastructure of eosinophilic inclusion bodies in the amygdala-parahippocampal region of aged squirrel monkeys treated with 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine, a dopaminergic neurotoxin.
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[Lewy bodies, a misleading marker for Parkinson's disease?].[路易小体，帕金森病的一个误导性标志物？]
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The spatial patterns of Lewy bodies, senile plaques, and neurofibrillary tangles in dementia with Lewy bodies.路易体痴呆中路易体、老年斑和神经原纤维缠结的空间分布模式。
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Pro-apoptotic protein glyceraldehyde-3-phosphate dehydrogenase promotes the formation of Lewy body-like inclusions.促凋亡蛋白甘油醛-3-磷酸脱氢酶促进路易小体样包涵体的形成。
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Temporal lobe atrophy on MRI in Parkinson disease with dementia: a comparison with Alzheimer disease and dementia with Lewy bodies.帕金森病痴呆患者MRI上的颞叶萎缩：与阿尔茨海默病和路易体痴呆的比较
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Development of alpha-synuclein immunoreactive astrocytes in the forebrain parallels stages of intraneuronal pathology in sporadic Parkinson's disease.在前脑，α-突触核蛋白免疫反应性星形胶质细胞的发育与散发性帕金森病中神经元内病理学阶段平行。
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Cortical and amygdalar Lewy body burden in Parkinson's disease patients with visual hallucinations.帕金森病伴视幻觉患者的皮质和杏仁核路易小体负荷
Parkinsonism Relat Disord. 2006 May;12(4):253-6. doi: 10.1016/j.parkreldis.2005.10.005. Epub 2005 Dec 20.

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Defining a Lewy Body: Running Up the Hill of Shifting Definitions and Evolving Concepts.定义路易小体：在不断变化的定义和发展的概念之山上攀登。
J Parkinsons Dis. 2024;14(1):17-33. doi: 10.3233/JPD-230183.
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The Role of the Thioredoxin System in Brain Diseases.硫氧还蛋白系统在脑部疾病中的作用。
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Revisit the Cellular Transmission and Emerging Techniques in Understanding the Mechanisms of Proteinopathies.重新审视细胞传递及新兴技术以理解蛋白质病的发病机制。
Front Neurosci. 2021 Nov 18;15:781722. doi: 10.3389/fnins.2021.781722. eCollection 2021.
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The process of Lewy body formation, rather than simply α-synuclein fibrillization, is one of the major drivers of neurodegeneration.路易体的形成过程，而不仅仅是α-突触核蛋白的纤维化，是神经退行性变的主要驱动因素之一。
Proc Natl Acad Sci U S A. 2020 Mar 3;117(9):4971-4982. doi: 10.1073/pnas.1913904117. Epub 2020 Feb 19.
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Neuropathology and pathogenesis of extrapyramidal movement disorders: a critical update-I. Hypokinetic-rigid movement disorders.锥体外系运动障碍的神经病理学和发病机制：批判性更新-I. 运动减少-僵硬的运动障碍。
J Neural Transm (Vienna). 2019 Aug;126(8):933-995. doi: 10.1007/s00702-019-02028-6. Epub 2019 Jun 18.
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Mitochondrial mechanisms of redox cycling agents implicated in Parkinson's disease.与帕金森病相关的氧化还原循环剂的线粒体机制。
J Neural Transm (Vienna). 2016 Feb;123(2):113-23. doi: 10.1007/s00702-015-1386-4. Epub 2015 Mar 7.
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Interplay between sumoylation and phosphorylation for protection against α-synuclein inclusions.泛素化和磷酸化之间的相互作用可防止α-突触核蛋白包涵体的形成。
J Biol Chem. 2014 Nov 7;289(45):31224-40. doi: 10.1074/jbc.M114.559237. Epub 2014 Sep 17.
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Aggregate clearance of α-synuclein in Saccharomyces cerevisiae depends more on autophagosome and vacuole function than on the proteasome.α-突触核蛋白在酿酒酵母中的总体清除率更依赖于自噬体和液泡功能，而不是蛋白酶体。
J Biol Chem. 2012 Aug 10;287(33):27567-79. doi: 10.1074/jbc.M112.361865. Epub 2012 Jun 21.
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Postmortem studies in Parkinson's disease.帕金森病的尸检研究。
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Increased iron levels correlate with the selective nigral dopaminergic neuron degeneration in Parkinson's disease.铁含量升高与帕金森病中选择性黑质多巴胺能神经元变性相关。
J Neural Transm (Vienna). 2011 Mar;118(3):361-9. doi: 10.1007/s00702-010-0434-3. Epub 2010 Jun 17.