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Towards a definition of the HCMV entry pathway.

作者信息

Compton T

机构信息

Department of Medical Microbiology and Immunology, University of Wisconsin, Madison Medical School, USA.

出版信息

Scand J Infect Dis Suppl. 1995;99:30-2.

PMID:8668939
Abstract
摘要

相似文献

1
Towards a definition of the HCMV entry pathway.
Scand J Infect Dis Suppl. 1995;99:30-2.
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Hepatocytes are permissive for human cytomegalovirus infection in human liver cell culture and In vivo.在人肝细胞培养物和体内,肝细胞对人巨细胞病毒感染具有易感性。
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Manipulation of autophagy by HCMV infection is involved in mTOR and influences the replication of virus.人巨细胞病毒感染对自噬的调控涉及雷帕霉素靶蛋白,并影响病毒复制。
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Entry of human cytomegalovirus into retinal pigment epithelial and endothelial cells by endocytosis.人巨细胞病毒通过内吞作用进入视网膜色素上皮细胞和内皮细胞。
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Platelet-derived growth factor-alpha receptor activation is required for human cytomegalovirus infection.人巨细胞病毒感染需要血小板衍生生长因子-α受体激活。
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Postattachment events associated with viral entry are necessary for induction of interferon-stimulated genes by human cytomegalovirus.
与病毒进入相关的附着后事件对于人巨细胞病毒诱导干扰素刺激基因是必需的。
J Virol. 2004 Jun;78(12):6688-91. doi: 10.1128/JVI.78.12.6688-6691.2004.
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Characterization of a panel of insertion mutants in human cytomegalovirus glycoprotein B.一组人巨细胞病毒糖蛋白B插入突变体的特性分析
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Mol Cell Biol. 1999 May;19(5):3607-13. doi: 10.1128/MCB.19.5.3607.
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The human cytomegalovirus UL55 (gB) and UL75 (gH) glycoprotein ligands initiate the rapid activation of Sp1 and NF-kappaB during infection.人巨细胞病毒UL55(gB)和UL75(gH)糖蛋白配体在感染过程中引发Sp1和核因子κB的快速激活。
J Virol. 1997 Jul;71(7):5051-9. doi: 10.1128/JVI.71.7.5051-5059.1997.