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[家鼠(小家鼠)切尔诺贝利核电站事故的遗传后遗症]

[Genetic sequelae of the accident at the Chernobyl nuclear power plant in house mice (Mus musculus)].

作者信息

Pomerantseva M D, Ramaĭa L K, Chekhovich A V

出版信息

Genetika. 1996 Feb;32(2):298-303.

PMID:8713627
Abstract

Genetic disorders were studied in house mice caught from 1986 to 1993 in areas contaminated by radionuclides after the Chernobyl disaster. Dose rates on soil surface ranged from 0.02 to 200 mR/h. Frequency of reciprocal translocations in spermatocytes of the mice studied was relatively low, but increased with dose rate. In populations, frequency of mice heterozygous for recessive lethal mutations decreased with time after the accident. The data obtained allow us to assume that induced mutations may lead to elimination of germ cells and decreased viability in mice heterozygous for the mutations. These processes result in removing excess mutations from the population.

摘要

对1986年至1993年在切尔诺贝利灾难后受放射性核素污染地区捕获的家鼠进行了遗传疾病研究。土壤表面的剂量率范围为0.02至200毫伦琴/小时。所研究小鼠精母细胞中相互易位的频率相对较低,但随剂量率增加。在种群中,隐性致死突变杂合子小鼠的频率在事故发生后随时间下降。所获得的数据使我们能够假设,诱导突变可能导致生殖细胞的消除以及突变杂合子小鼠的生存能力下降。这些过程导致种群中多余突变的清除。

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