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The epidermal hyperplasia associated with repeated barrier disruption by acetone treatment or tape stripping cannot be attributed to increased water loss.

作者信息

Denda M, Wood L C, Emami S, Calhoun C, Brown B E, Elias P M, Feingold K R

机构信息

Department of Medicine, University of California, San Francisco, USA.

出版信息

Arch Dermatol Res. 1996 May;288(5-6):230-8. doi: 10.1007/BF02530090.

DOI:10.1007/BF02530090
PMID:8738565
Abstract
摘要

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The epidermal hyperplasia associated with repeated barrier disruption by acetone treatment or tape stripping cannot be attributed to increased water loss.与丙酮处理或胶带剥离反复破坏屏障相关的表皮增生不能归因于水分流失增加。
Arch Dermatol Res. 1996 May;288(5-6):230-8. doi: 10.1007/BF02530090.
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trans-4-(Aminomethyl)cyclohexane carboxylic acid (T-AMCHA), an anti-fibrinolytic agent, accelerates barrier recovery and prevents the epidermal hyperplasia induced by epidermal injury in hairless mice and humans.
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Barium sulphate with a negative zeta potential accelerates skin permeability barrier recovery and prevents epidermal hyperplasia induced by barrier disruption.具有负ζ电位的硫酸钡可加速皮肤渗透屏障的恢复，并防止屏障破坏引起的表皮增生。
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Topical stratum corneum lipids accelerate barrier repair after tape stripping, solvent treatment and some but not all types of detergent treatment.外用角质层脂质可加速胶带剥离、溶剂处理以及部分（而非全部）类型的去污剂处理后的屏障修复。
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NMDA-type glutamate receptor is associated with cutaneous barrier homeostasis.N-甲基-D-天冬氨酸（NMDA）型谷氨酸受体与皮肤屏障稳态相关。
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Effect of endothelial nitric oxide synthase on epidermal permeability barrier recovery after disruption.内皮型一氧化氮合酶对表皮屏障破坏后恢复的影响。
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Establishment of an atopic dermatitis-like skin model in a hairless mouse by repeated elicitation of contact hypersensitivity that enables to conduct functional analyses of the stratum corneum with various non-invasive biophysical instruments.通过反复激发接触性超敏反应在无毛小鼠中建立特应性皮炎样皮肤模型，该模型能够使用各种非侵入性生物物理仪器对角质层进行功能分析。
Skin Res Technol. 2004 May;10(2):122-9. doi: 10.1111/j.1600-0846.2004.00062.x.

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Oral Intake of Linseed Oil Inhibits Skin Barrier Dysfunction in Obese Mice.口服亚麻籽油可抑制肥胖小鼠的皮肤屏障功能障碍。

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1
Barrier function regulates epidermal lipid and DNA synthesis.屏障功能调节表皮脂质和DNA合成。
Br J Dermatol. 1993 May;128(5):473-82. doi: 10.1111/j.1365-2133.1993.tb00222.x.
2
Processing of epidermal glucosylceramides is required for optimal mammalian cutaneous permeability barrier function.最佳的哺乳动物皮肤渗透屏障功能需要表皮葡糖神经酰胺的加工过程。
J Clin Invest. 1993 Apr;91(4):1656-64. doi: 10.1172/JCI116374.
3
Selective obliteration of the epidermal calcium gradient leads to enhanced lamellar body secretion.选择性消除表皮钙梯度会导致板层小体分泌增加。
Cureus. 2024 May 30;16(5):e61392. doi: 10.7759/cureus.61392. eCollection 2024 May.
4
"Outside-to-inside," "inside-to-outside," and "intrinsic" endogenous pathogenic mechanisms in atopic dermatitis: keratinocytes as the key functional cells involved in both permeability barrier dysfunction and immunological alterations.特应性皮炎的“外-内”、“内-外”和“内在”内源性发病机制：角朊细胞作为涉及渗透性屏障功能障碍和免疫改变的关键功能细胞。
Front Immunol. 2023 Aug 11;14:1239251. doi: 10.3389/fimmu.2023.1239251. eCollection 2023.
5
Optimizing emollient therapy for skin barrier repair in atopic dermatitis.优化保湿剂治疗特应性皮炎的皮肤屏障修复。
Ann Allergy Asthma Immunol. 2022 May;128(5):505-511. doi: 10.1016/j.anai.2022.01.012. Epub 2022 Jan 20.
6
Keratin 14-dependent disulfides regulate epidermal homeostasis and barrier function via 14-3-3σ and YAP1.角蛋白 14 依赖性二硫键通过 14-3-3σ 和 YAP1 调节表皮稳态和屏障功能。
Elife. 2020 May 5;9:e53165. doi: 10.7554/eLife.53165.
7
Stress test of the skin: The cutaneous permeability barrier treadmill.皮肤压力测试：皮肤渗透性屏障跑步机。
Exp Dermatol. 2020 Jan;29(1):112-113. doi: 10.1111/exd.14055. Epub 2019 Nov 22.
8
Does moisturizing the skin equate with barrier repair therapy?皮肤保湿等同于屏障修复疗法吗？
Ann Allergy Asthma Immunol. 2018 Dec;121(6):653-656.e2. doi: 10.1016/j.anai.2018.07.008. Epub 2018 Aug 10.
9
Reduced skin lipid content in obese Japanese women mediated by decreased expression of rate-limiting lipogenic enzymes.肥胖日本女性皮肤脂质含量降低，由限速脂肪生成酶表达减少介导。
PLoS One. 2018 Mar 8;13(3):e0193830. doi: 10.1371/journal.pone.0193830. eCollection 2018.
10
Analysis of gene expression dynamics revealed delayed and abnormal epidermal repair process in aged compared to young skin.基因表达动力学分析显示，与年轻皮肤相比，老年皮肤的表皮修复过程延迟且异常。
Arch Dermatol Res. 2015 May;307(4):351-64. doi: 10.1007/s00403-015-1551-5. Epub 2015 Mar 6.
J Invest Dermatol. 1994 May;102(5):789-95. doi: 10.1111/1523-1747.ep12377921.
4
Integrity of the permeability barrier is crucial for maintenance of the epidermal calcium gradient.渗透屏障的完整性对于维持表皮钙梯度至关重要。
Br J Dermatol. 1994 Feb;130(2):139-47. doi: 10.1111/j.1365-2133.1994.tb02892.x.
5
Permeability barrier disruption alters the localization and expression of TNF alpha/protein in the epidermis.
Arch Dermatol Res. 1994;286(5):242-8. doi: 10.1007/BF00387595.
6
Occlusion lowers cytokine mRNA levels in essential fatty acid-deficient and normal mouse epidermis, but not after acute barrier disruption.在必需脂肪酸缺乏和正常的小鼠表皮中，封闭可降低细胞因子mRNA水平，但在急性屏障破坏后则不然。
J Invest Dermatol. 1994 Dec;103(6):834-8. doi: 10.1111/1523-1747.ep12413597.
7
Perturbation of epidermal barrier function correlates with initiation of cytokine cascade in human skin.表皮屏障功能的紊乱与人体皮肤中细胞因子级联反应的启动相关。
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Mitotic index of psoriatic lesions treated with anthralin, glucocorticosteriod and occlusion only.仅用蒽林、糖皮质激素和封包治疗的银屑病皮损的有丝分裂指数。
J Invest Dermatol. 1970 May;54(5):410-2. doi: 10.1111/1523-1747.ep12259209.
9
Effect of plastic occlusive dressings on psoriatic epidermis.塑料封闭敷料对银屑病表皮的影响。
Br J Dermatol. 1970 May;82(5):458-62. doi: 10.1111/j.1365-2133.1970.tb02206.x.
10
Research into psoriasis--the last decade.银屑病研究——过去十年
Br Med J. 1971 Jul 24;3(5768):236-9. doi: 10.1136/bmj.3.5768.236.