紫外线照射后，着色性干皮病A互补组细胞中p53的核内积累时间延长。 - Suppr

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超能文献

Prolonged nuclear accumulation of p53 in xeroderma pigmentosum complementation group A cells after ultraviolet irradiation.

作者信息

Kato S, Urano Y, Sasaki S, Ahsan K, Shono M, Arase S

机构信息

Department of Dermatology, School of Medicine, University of Tokushima, Japan.

出版信息

Arch Dermatol Res. 1996 May;288(5-6):249-51. doi: 10.1007/BF02530093.

DOI:10.1007/BF02530093

PMID:8738568

Abstract

摘要

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Prolonged nuclear accumulation of p53 in xeroderma pigmentosum complementation group A cells after ultraviolet irradiation.紫外线照射后，着色性干皮病A互补组细胞中p53的核内积累时间延长。

Arch Dermatol Res. 1996 May;288(5-6):249-51. doi: 10.1007/BF02530093.

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本文引用的文献

Thymocyte apoptosis induced by p53-dependent and independent pathways.由p53依赖和非依赖途径诱导的胸腺细胞凋亡。

Nature. 1993 Apr 29;362(6423):849-52. doi: 10.1038/362849a0.

p53 is required for radiation-induced apoptosis in mouse thymocytes.p53是小鼠胸腺细胞辐射诱导凋亡所必需的。

Nature. 1993 Apr 29;362(6423):847-9. doi: 10.1038/362847a0.

Induction of nuclear accumulation of the tumor-suppressor protein p53 by DNA-damaging agents.DNA损伤剂诱导肿瘤抑制蛋白p53的核内聚集。

Oncogene. 1993 Feb;8(2):307-18.

Aerobic responses of mouse macrophages to phagocytosis against Escherichia coli as revealed by microphotometry.显微光度法揭示的小鼠巨噬细胞对吞噬大肠杆菌的需氧反应。

Cell Mol Biol (Noisy-le-grand). 1993 Jun;39(4):361-70.

Differential induction of transcriptionally active p53 following UV or ionizing radiation: defects in chromosome instability syndromes?紫外线或电离辐射后转录活性p53的差异诱导：染色体不稳定综合征中的缺陷？

Cell. 1993 Nov 19;75(4):765-78. doi: 10.1016/0092-8674(93)90496-d.

p53 gene mutations in human skin cancers and precancerous lesions: comparison with immunohistochemical analysis.人类皮肤癌和癌前病变中的p53基因突变：与免疫组织化学分析的比较

J Invest Dermatol. 1994 Apr;102(4):440-4. doi: 10.1111/1523-1747.ep12373002.

DNA strand breaks: the DNA template alterations that trigger p53-dependent DNA damage response pathways.DNA链断裂：触发p53依赖性DNA损伤反应途径的DNA模板改变。

Mol Cell Biol. 1994 Mar;14(3):1815-23. doi: 10.1128/mcb.14.3.1815-1823.1994.

U.v.-induced nuclear accumulation of p53 is evoked through DNA damage of actively transcribed genes independent of the cell cycle.紫外线诱导的p53核积累是通过活跃转录基因的DNA损伤引发的，与细胞周期无关。

Oncogene. 1994 Oct;9(10):2775-84.

Sensitivity to UV radiation of fibroblasts from a Japanese group A xeroderma pigmentosum patient with mild neurological abnormalities.一名患有轻度神经异常的日本色素性干皮病 A 组患者的成纤维细胞对紫外线辐射的敏感性。

Br J Dermatol. 1987 Jan;116(1):101-8. doi: 10.1111/j.1365-2133.1987.tb05796.x.

Xeroderma pigmentosum. Cutaneous, ocular, and neurologic abnormalities in 830 published cases.着色性干皮病。830例已发表病例中的皮肤、眼部及神经异常。

Arch Dermatol. 1987 Feb;123(2):241-50. doi: 10.1001/archderm.123.2.241.

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