尽管受到紫外线B照射，但来自干扰素γ处理过的人类皮肤的MHC II类角质形成细胞在存在葡萄球菌超抗原的情况下仍能激活T细胞。 - Suppr

Suppr

超能文献

MHC class II+ keratinocytes from IFN gamma-treated human skin activate T cells in the presence of staphylococcal superantigen despite UVB irradiation.

作者信息

Skov L, Baadsgaard O

机构信息

Department of Dermatology, Gentofte Hospital, Hellerup, Denmark.

出版信息

Arch Dermatol Res. 1996 May;288(5-6):255-7. doi: 10.1007/BF02530095.

DOI:10.1007/BF02530095

PMID:8738570

Abstract

摘要

相似文献

MHC class II+ keratinocytes from IFN gamma-treated human skin activate T cells in the presence of staphylococcal superantigen despite UVB irradiation.尽管受到紫外线B照射，但来自干扰素γ处理过的人类皮肤的MHC II类角质形成细胞在存在葡萄球菌超抗原的情况下仍能激活T细胞。

Arch Dermatol Res. 1996 May;288(5-6):255-7. doi: 10.1007/BF02530095.

Interferon gamma-treated keratinocytes activate T cells in the presence of superantigens: involvement of major histocompatibility complex class II molecules.γ干扰素处理的角质形成细胞在超抗原存在的情况下激活T细胞：主要组织相容性复合体II类分子的参与

J Invest Dermatol. 1994 Feb;102(2):150-4. doi: 10.1111/1523-1747.ep12371753.

Ultraviolet B-exposed major histocompatibility complex class ii positive keratinocytes and antigen-presenting cells demonstrate a differential capacity to activate T cells in the presence of staphylococcal superantigens.暴露于紫外线B的主要组织相容性复合体II类阳性角质形成细胞和抗原呈递细胞在存在葡萄球菌超抗原的情况下表现出不同的激活T细胞的能力。

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Superantigenic staphylococcal exotoxins induce T-cell proliferation in the presence of Langerhans cells or class II-bearing keratinocytes and stimulate keratinocytes to produce T-cell-activating cytokines.超抗原性葡萄球菌外毒素在朗格汉斯细胞或表达II类分子的角质形成细胞存在的情况下诱导T细胞增殖，并刺激角质形成细胞产生T细胞激活细胞因子。

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T-cell proliferation to superantigen-releasing Staphylococcus aureus by MHC class II-bearing keratinocytes under protection from bacterial cytolysin.在免受细菌溶细胞素影响的情况下，由表达MHC-II类分子的角质形成细胞介导的T细胞对释放超抗原的金黄色葡萄球菌的增殖反应

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Porcine MHC classical class I genes are coordinately expressed in superantigen-activated mononuclear cells.猪主要组织相容性复合体（MHC）经典I类基因在超抗原激活的单核细胞中协同表达。

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Accessory cell function of keratinocytes for superantigens. Dependence on lymphocyte function-associated antigen-1/intercellular adhesion molecule-1 interaction.角质形成细胞对超抗原的辅助细胞功能。依赖淋巴细胞功能相关抗原-1/细胞间黏附分子-1相互作用。

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Control of T cell responses to staphylococcal enterotoxins by stimulator cell MHC class II polymorphism.刺激细胞MHC II类多态性对T细胞对葡萄球菌肠毒素反应的调控。

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本文引用的文献

Superantigenic exotoxin-secreting potential of staphylococci isolated from atopic eczematous skin.

Br J Dermatol. 1993 Jun;128(6):631-2. doi: 10.1111/j.1365-2133.1993.tb00257.x.

Streptococcal and staphylococcal superantigens (ETA, SEB): presentation by human epidermal cells and induction of autologous T cell proliferation in vitro.

Acta Derm Venereol. 1993 Apr;73(2):94-6. doi: 10.2340/00015555739496.

J Invest Dermatol. 1994 Feb;102(2):150-4. doi: 10.1111/1523-1747.ep12371753.

Evidence of HLA-DR antigen biosynthesis by human keratinocytes in disease.人类角质形成细胞在疾病中进行HLA - DR抗原生物合成的证据。

J Exp Med. 1984 Jun 1;159(6):1784-9. doi: 10.1084/jem.159.6.1784.

Staphylococcus aureus in psoriasis.

Arch Dermatol. 1973 Apr;107(4):568-70.

Staphylococcus aureus in the lesions of atopic dermatitis.特应性皮炎皮损中的金黄色葡萄球菌。

Br J Dermatol. 1974 May;90(5):525-30. doi: 10.1111/j.1365-2133.1974.tb06447.x.

Effects of ultraviolet radiation on human epidermal cell alloantigen presentation: initial depression of Langerhans cell-dependent function is followed by the appearance of T6- Dr+ cells that enhance epidermal alloantigen presentation.紫外线辐射对人表皮细胞同种异体抗原呈递的影响：朗格汉斯细胞依赖性功能最初受到抑制，随后出现增强表皮同种异体抗原呈递的T6-Dr+细胞。

J Immunol. 1985 Jan;134(1):129-37.

Effect of UVB on alloactivating and antigen-presenting capacity of human epidermal Langerhans cells.

Scand J Immunol. 1985 May;21(5):417-23. doi: 10.1111/j.1365-3083.1985.tb01827.x.

Antigen-presenting OKM5+ melanophages appear in human epidermis after ultraviolet radiation.紫外线照射后，抗原呈递性OKM5 +黑素巨噬细胞出现在人类表皮中。

J Invest Dermatol. 1986 Apr;86(4):363-70. doi: 10.1111/1523-1747.ep12285600.

Rapid recovery of Langerhans cell alloreactivity, without induction of autoreactivity, after in vivo ultraviolet A, but not ultraviolet B exposure of human skin.人体皮肤经体内紫外线A而非紫外线B照射后，朗格汉斯细胞同种异体反应性迅速恢复，且未诱导自身反应性。

J Immunol. 1989 Jun 15;142(12):4213-8.

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