顺铂诱导培养的人角质形成细胞凋亡时Fas抗原表达上调。 - Suppr

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超能文献

Upregulated expression of Fas antigen on cultured human keratinocytes with induction of apoptosis by cisplatin.

作者信息

Matsumoto Y, Hayakawa A, Tamada Y, Mori H, Ohashi M

机构信息

Department of Dermatology, Nagoya University School of Medicine, Japan.

出版信息

Arch Dermatol Res. 1996 May;288(5-6):267-9. doi: 10.1007/BF02530099.

DOI:10.1007/BF02530099

PMID:8738574

Abstract

摘要

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Upregulated expression of Fas antigen on cultured human keratinocytes with induction of apoptosis by cisplatin.顺铂诱导培养的人角质形成细胞凋亡时Fas抗原表达上调。

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本文引用的文献

Apoptosis in the pathogenesis and treatment of disease.细胞凋亡在疾病的发病机制及治疗中的作用

Science. 1995 Mar 10;267(5203):1456-62. doi: 10.1126/science.7878464.

Fas and Fas ligand: lpr and gld mutations.Fas与Fas配体：lpr和gld突变

Immunol Today. 1995 Jan;16(1):39-43. doi: 10.1016/0167-5699(95)80069-7.

The Fas death factor.Fas死亡因子。

Science. 1995 Mar 10;267(5203):1449-56. doi: 10.1126/science.7533326.

Arch Dermatol Res. 1994;286(7):396-407. doi: 10.1007/BF00371800.

J Invest Dermatol. 1994 Sep;103(3):330-4. doi: 10.1111/1523-1747.ep12394858.

Tyrosine kinase activation provides an early and requisite signal for Fas-induced apoptosis.

J Immunol. 1994 Sep 1;153(5):1947-54.

Induction of endonucleolytic DNA cleavage in human acute myelogenous leukemia cells by etoposide, camptothecin, and other cytotoxic anticancer drugs: a cautionary note.依托泊苷、喜树碱及其他细胞毒性抗癌药物诱导人急性髓性白血病细胞发生核酸内切酶介导的DNA裂解：一则警示

Cancer Res. 1989 Nov 1;49(21):5870-8.

A cell-killing monoclonal antibody (anti-Fas) to a cell surface antigen co-downregulated with the receptor of tumor necrosis factor.一种针对与肿瘤坏死因子受体共同下调的细胞表面抗原的细胞杀伤性单克隆抗体（抗Fas）。

J Exp Med. 1989 May 1;169(5):1747-56. doi: 10.1084/jem.169.5.1747.

Activation of programmed cell death by anticancer agents: cisplatin as a model system.抗癌药物诱导的程序性细胞死亡：以顺铂作为模型系统

Cancer Cells. 1990 Aug-Sep;2(8-9):275-80.

The polypeptide encoded by the cDNA for human cell surface antigen Fas can mediate apoptosis.

Cell. 1991 Jul 26;66(2):233-43. doi: 10.1016/0092-8674(91)90614-5.

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