Cohen M, Luxon L, Rudge P
Division of Audiological Medicine, University College London, UK.
Scand Audiol. 1996;25(2):133-41. doi: 10.3109/01050399609047995.
Four cases of central pontine haemorrhage are described in which auditory dysfunction was documented. Two cases had a hearing loss, in one of which there was recovery of the low frequencies. This case provides support for the tonotopic organization of the auditory pathways in the caudal pontine area, with the lowest frequencies being encoded medially. In all cases, there were abnormalities of the auditory brainstem responses, wave V being consistently involved, while wave III was abnormal in only one patient. In three cases, the masking level differences and crossed acoustic reflex thresholds were abnormal. The ipsilateral reflex thresholds were normal at least on one side in all cases. In the patient with the most significant hearing loss, loudness recruitment, assessed both psychophysically and with the acoustic reflex thresholds, was evident. These data are interpreted in terms of there being damage to the medial superior olivary nuclei and trapezoid body involving both afferent and efferent fibres.
本文描述了4例记录有听觉功能障碍的桥脑出血病例。2例存在听力损失，其中1例低频听力有所恢复。该病例支持了脑桥尾侧区听觉通路的音频定位组织，最低频率在内侧编码。所有病例中，听觉脑干反应均有异常，V波均持续受累，而只有1例患者的III波异常。3例病例的掩蔽级差和交叉听觉反射阈值异常。所有病例中，同侧反射阈值至少在一侧是正常的。在听力损失最严重的患者中，通过心理物理学和听觉反射阈值评估均发现明显的响度重振现象。这些数据被解释为内侧上橄榄核和斜方体受损，涉及传入和传出纤维。
