Enhancement of intestinal glucose transport following chronic parathion poisoning.

Chronic administration of parathion to male rats stimulated glucose (p less than 0.05) but not calcium (p greater than 0.05) transport in the everted duodenal gut sac preparation. Chronic parathion stimulation was not reduced by concurrent administered of atropine. Acutely applied parathion or paraoxon, its active metabolite, did not increase glucose transport in this preparation.