慢性对硫磷中毒后肠道葡萄糖转运增强。
Enhancement of intestinal glucose transport following chronic parathion poisoning.
作者信息
Robinson C P, Choi J J, Pento J T
出版信息
J Pharm Sci. 1977 Jun;66(6):879-80. doi: 10.1002/jps.2600660640.
PMID:874796
Abstract
Chronic administration of parathion to male rats stimulated glucose (p less than 0.05) but not calcium (p greater than 0.05) transport in the everted duodenal gut sac preparation. Chronic parathion stimulation was not reduced by concurrent administered of atropine. Acutely applied parathion or paraoxon, its active metabolite, did not increase glucose transport in this preparation.
摘要
对雄性大鼠长期施用对硫磷会刺激外翻十二指肠肠囊制剂中的葡萄糖转运（p<0.05），但不会刺激钙转运（p>0.05）。同时施用阿托品并不会降低对硫磷的长期刺激作用。急性施用对硫磷或其活性代谢产物对氧磷，并不会增加该制剂中的葡萄糖转运。
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