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Store-operated Ca2+ influx: what is the message from the stores to the membrane?

作者信息

Favre C J, Nüsse O, Lew D P, Krause K H

机构信息

Division of Infectious Diseases, University Hospital, Geneva, Switzerland.

出版信息

J Lab Clin Med. 1996 Jul;128(1):19-26. doi: 10.1016/s0022-2143(96)90110-9.

DOI:10.1016/s0022-2143(96)90110-9
PMID:8759933
Abstract
摘要

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The FPR2-induced rise in cytosolic calcium in human neutrophils relies on an emptying of intracellular calcium stores and is inhibited by a gelsolin-derived PIP2-binding peptide.甲酰肽受体2（FPR2）诱导的人中性粒细胞胞质钙升高依赖于细胞内钙库排空，并受到凝溶胶蛋白衍生的磷脂酰肌醇-4,5-二磷酸（PIP2）结合肽的抑制。
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Pharmacological control of neutrophil-mediated inflammation: strategies targeting calcium handling by activated polymorphonuclear leukocytes.中性粒细胞介导的炎症的药理学控制：针对活化多形核白细胞钙处理的策略。
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Taming the neutrophil: calcium clearance and influx mechanisms as novel targets for pharmacological control.驯服中性粒细胞：钙清除与内流机制作为药理学控制的新靶点
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Infect Immun. 1999 Oct;67(10):5157-62. doi: 10.1128/IAI.67.10.5157-5162.1999.