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Recovery of behavioral functions following the transplantation of the embryonal striatum into the damaged amygdala of the rat brain.

作者信息

Ermakova I V, Zhulin V V

机构信息

Laboratory of Compensatory Brain Functions:, Russian Academy of Sciences, Moscow.

出版信息

Neurosci Behav Physiol. 1996 Jan-Feb;26(1):11-2. doi: 10.1007/BF02391150.

DOI:10.1007/BF02391150
PMID:8801463
Abstract
摘要

相似文献

1
Recovery of behavioral functions following the transplantation of the embryonal striatum into the damaged amygdala of the rat brain.将胚胎纹状体移植到大鼠脑损伤杏仁核后行为功能的恢复
Neurosci Behav Physiol. 1996 Jan-Feb;26(1):11-2. doi: 10.1007/BF02391150.
2
[The recovery of behavioral functions after the transplantation of embryonic striatum to the damaged amygdala in rats].[大鼠胚胎纹状体移植至受损杏仁核后行为功能的恢复]
Fiziol Zh Im I M Sechenova. 1994 Apr;80(4):113-5.
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Irreversibility of disruptions of integrative activity induced by damage to the amygdala, with unilateral transplantation of embryonal amygdalar tissue.杏仁核损伤所致整合活动破坏的不可逆性，伴有胚胎杏仁核组织的单侧移植。
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Glial cell line-derived neurotrophic factor improves intrastriatal graft survival of stored dopaminergic cells.胶质细胞系源性神经营养因子可提高储存多巴胺能细胞纹状体内移植的存活率。
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Development of long-distance efferent projections from fetal hippocampal grafts depends upon pathway specificity and graft location in kainate-lesioned adult hippocampus.胎儿海马移植体的长距离传出投射的发育取决于在成年海马体经红藻氨酸损伤后的通路特异性和移植体位置。
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Significance of the character and probability of reinforcement in animals with transplantation of various embryonal tissue structures.不同胚胎组织结构移植动物中强化的特征及可能性的意义
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Fetal mesencephalic grafts influence the dopamine release in the non-lesioned striatum of 6-OHDA-lesioned rats: a behavioral and in vivo voltammetric study.
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Effects of graft pooling of foetal rat and mouse tissue and immunosuppression in the 6-hydroxydopamine rat model of Parkinson's disease.胎儿大鼠和小鼠组织移植合并及免疫抑制在帕金森病6-羟基多巴胺大鼠模型中的作用
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Assessment of fetal tissue transplantation in Parkinson's disease: does PET play a role?帕金森病中胎儿组织移植的评估：正电子发射断层扫描（PET）有作用吗？
Neurology. 1994 Oct;44(10):1777-80. doi: 10.1212/wnl.44.10.1777.

本文引用的文献

1
Irrelevant behaviour, information processing and arousal homeostasis.无关行为、信息处理与唤醒稳态。
Psychol Forsch. 1970;33(2):165-88. doi: 10.1007/BF00424983.
2
Kainic acid: a powerful neurotoxic analogue of glutamate.海人酸：一种强大的谷氨酸神经毒性类似物。
Brain Res. 1974 Sep 13;77(3):507-12. doi: 10.1016/0006-8993(74)90640-4.
3
Locomotor hyperactivity: effects of multiple striatal transplants in an animal model of Huntington's disease.运动性多动：亨廷顿舞蹈病动物模型中多次纹状体移植的影响
Pharmacol Biochem Behav. 1986 Jul;25(1):297-300. doi: 10.1016/0091-3057(86)90269-8.
4
Limbic seizure and brain damage produced by kainic acid: mechanisms and relevance to human temporal lobe epilepsy.海藻酸诱发的边缘叶癫痫发作和脑损伤：机制及其与人类颞叶癫痫的相关性
Neuroscience. 1985 Feb;14(2):375-403. doi: 10.1016/0306-4522(85)90299-4.
5
The effect of embryonal amygdala grafts on the impairment of spatial working memory elicited in rats by kainate-induced amygdaloid damage.胚胎杏仁核移植对红藻氨酸诱导的杏仁核损伤所致大鼠空间工作记忆损害的影响。
Physiol Behav. 1989 Feb;45(2):235-41. doi: 10.1016/0031-9384(89)90124-8.
6
Limbic seizures without brain damage after injection of low doses of kainic acid into the amygdala of freely moving rats.在自由活动的大鼠杏仁核中注射低剂量海人酸后出现无脑损伤的边缘叶癫痫发作。
Brain Res. 1989 Jun 12;489(2):261-72. doi: 10.1016/0006-8993(89)90859-7.