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Biological properties of VEGF/VPF receptors.

作者信息

Terman B I, Dougher-Vermazen M

机构信息

Wyeth-Ayerst Oncology Research, Pearl River, NY 10965, USA.

出版信息

Cancer Metastasis Rev. 1996 Jun;15(2):159-63. doi: 10.1007/BF00437468.

DOI:10.1007/BF00437468
PMID:8842487
Abstract
摘要

相似文献

1
Biological properties of VEGF/VPF receptors.
Cancer Metastasis Rev. 1996 Jun;15(2):159-63. doi: 10.1007/BF00437468.
2
Vascular endothelial growth factor.血管内皮生长因子
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3
Cartilage to bone--angiogenesis leads the way.软骨到骨——血管生成起主导作用。
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4
The role of vascular endothelial growth factor in angiogenesis.血管内皮生长因子在血管生成中的作用。
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5
Strong expression of kinase insert domain-containing receptor, a vascular permeability factor/vascular endothelial growth factor receptor in AIDS-associated Kaposi's sarcoma and cutaneous angiosarcoma.含激酶插入结构域受体（一种血管通透性因子/血管内皮生长因子受体）在艾滋病相关卡波西肉瘤和皮肤血管肉瘤中呈强表达。
Am J Pathol. 1996 Apr;148(4):1065-74.
6
Hypoxia-induced paracrine regulation of vascular endothelial growth factor receptor expression.缺氧诱导的血管内皮生长因子受体表达的旁分泌调节
J Clin Invest. 1996 Jan 15;97(2):469-76. doi: 10.1172/JCI118437.
7
Vascular permeability factor/vascular endothelial growth factor and the significance of microvascular hyperpermeability in angiogenesis.血管通透性因子/血管内皮生长因子及微血管高通透性在血管生成中的意义
Curr Top Microbiol Immunol. 1999;237:97-132. doi: 10.1007/978-3-642-59953-8_6.
8
Regulation of angiogenesis via vascular endothelial growth factor receptors.通过血管内皮生长因子受体对血管生成的调节。
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9
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10
The neurotransmitter dopamine inhibits angiogenesis induced by vascular permeability factor/vascular endothelial growth factor.神经递质多巴胺可抑制血管通透性因子/血管内皮生长因子诱导的血管生成。
Nat Med. 2001 May;7(5):569-74. doi: 10.1038/87895.

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Dihydroartemisinin targets VEGFR2 via the NF-κB pathway in endothelial cells to inhibit angiogenesis.双氢青蒿素通过内皮细胞中的核因子κB通路靶向血管内皮生长因子受体2以抑制血管生成。
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本文引用的文献

1
An essential heparin-binding domain in the fibroblast growth factor receptor kinase.成纤维细胞生长因子受体激酶中的一个必需肝素结合结构域。
Science. 1993 Mar 26;259(5103):1918-21. doi: 10.1126/science.8456318.
2
The FLT4 gene encodes a transmembrane tyrosine kinase related to the vascular endothelial growth factor receptor.FLT4基因编码一种与血管内皮生长因子受体相关的跨膜酪氨酸激酶。
Oncogene. 1993 May;8(5):1233-40.
3
Specificity of vascular endothelial cell growth factor receptor ligand binding domains.血管内皮细胞生长因子受体配体结合域的特异性
印楝素在两种乳腺癌细胞系中抑制侵袭和迁移，并下调尿激酶型纤溶酶原激活物受体趋化因子基因表达。
Cell Prolif. 2014 Dec;47(6):540-52. doi: 10.1111/cpr.12148.
4
The role of vascular actors in two dimensional dialogue of human bone marrow stromal cell and endothelial cell for inducing self-assembled network.血管因子在诱导人骨髓基质细胞和内皮细胞自组装网络的二维对话中的作用。
PLoS One. 2011 Feb 3;6(2):e16767. doi: 10.1371/journal.pone.0016767.
5
Strength and weakness of phase I to IV trials, with an emphasis on translational aspects.I至IV期试验的优势与不足，重点关注转化医学方面。
Breast Cancer Res. 2008;10 Suppl 4(Suppl 4):S22. doi: 10.1186/bcr2182. Epub 2008 Dec 18.
6
A vascular gene trap screen defines RasGRP3 as an angiogenesis-regulated gene required for the endothelial response to phorbol esters.一项血管基因捕获筛选将RasGRP3定义为一种血管生成调节基因，是内皮细胞对佛波酯反应所必需的。
Mol Cell Biol. 2004 Dec;24(24):10515-28. doi: 10.1128/MCB.24.24.10515-10528.2004.
7
Analysis of VEGF-responsive genes involved in the activation of endothelial cells.参与内皮细胞激活的VEGF反应基因分析。
Mol Cancer. 2003 Jul 9;2:25. doi: 10.1186/1476-4598-2-25.
8
Oligonucleotide NX1838 inhibits VEGF165-mediated cellular responses in vitro.寡核苷酸NX1838在体外抑制VEGF165介导的细胞反应。
In Vitro Cell Dev Biol Anim. 1999 Oct;35(9):533-42. doi: 10.1007/s11626-999-0064-y.
9
The rationale and future potential of angiogenesis inhibitors in neoplasia.肿瘤血管生成抑制剂的原理及未来潜力
Drugs. 1999 Jul;58(1):17-38. doi: 10.2165/00003495-199958010-00003.
10
Modulation of angiogenesis by human glioma xenograft models that differentially express vascular endothelial growth factor.通过差异表达血管内皮生长因子的人胶质瘤异种移植模型对血管生成进行调控。
Clin Exp Metastasis. 1998 Aug;16(6):559-68. doi: 10.1023/a:1006598419541.
Biochem Biophys Res Commun. 1994 May 30;201(1):326-30. doi: 10.1006/bbrc.1994.1705.
4
Heparin modulates the interaction of VEGF165 with soluble and cell associated flk-1 receptors.肝素调节血管内皮生长因子165（VEGF165）与可溶性及细胞相关的fms样酪氨酸激酶-1（flk-1）受体之间的相互作用。
J Biol Chem. 1994 Apr 29;269(17):12456-61.
5
Glioblastoma growth inhibited in vivo by a dominant-negative Flk-1 mutant.一种显性负性Flk-1突变体在体内抑制胶质母细胞瘤生长。
Nature. 1994 Feb 10;367(6463):576-9. doi: 10.1038/367576a0.
6
Biological activity and phosphorylation sites of the bacterially expressed cytosolic domain of the KDR VEGF-receptor.KDR血管内皮生长因子受体细菌表达的胞质结构域的生物活性和磷酸化位点
Biochem Biophys Res Commun. 1994 Nov 30;205(1):728-38. doi: 10.1006/bbrc.1994.2726.
7
Different signal transduction properties of KDR and Flt1, two receptors for vascular endothelial growth factor.血管内皮生长因子的两种受体KDR和Flt1的不同信号转导特性
J Biol Chem. 1994 Oct 28;269(43):26988-95.
8
Placenta growth factor. Potentiation of vascular endothelial growth factor bioactivity, in vitro and in vivo, and high affinity binding to Flt-1 but not to Flk-1/KDR.胎盘生长因子。在体内外增强血管内皮生长因子的生物活性，与Flt-1具有高亲和力结合，但与Flk-1/KDR无结合。
J Biol Chem. 1994 Oct 14;269(41):25646-54.
9
Overexpression of vascular permeability factor (VPF/VEGF) and its endothelial cell receptors in delayed hypersensitivity skin reactions.血管通透性因子（VPF/VEGF）及其内皮细胞受体在迟发型超敏反应性皮肤反应中的过表达。
J Immunol. 1995 Mar 15;154(6):2801-7.
10
A unique signal transduction from FLT tyrosine kinase, a receptor for vascular endothelial growth factor VEGF.一种源自FLT酪氨酸激酶（血管内皮生长因子VEGF的受体）的独特信号转导。
Oncogene. 1995 Jan 5;10(1):135-47.