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核心技术专利：CN118964589B侵权必究

核心技术专利：CN118964589B侵权必究

Nara Y, Ikeda K, Matsumoto C, Nabika T, Sawamura M, Yamori Y

Graduate School of Human and Environmental Studies, Kyoto University, Japan.

Clin Exp Pharmacol Physiol. 1995 Dec;22(12):S382-5.

Basal high blood pressure (BP) in the male and female F2 progeny produced by crosses between Wistar Kyoto rats (WKY/lzm) and stroke-prone spontaneously hypertensive rats (SHRSP/lzm) linked strongly with the leukosialin locus on chromosome 1. 2. Excess salt intake caused elevation of BP and the response was larger in the females than the males. 3. In the male F2 progeny, salt-sensitive hypertension significantly cosegregated with the RR1023 marker on chromosome 10, rather than with the angiotensin converting enzyme locus. 4. In the female F2 progeny, salt-sensitive hypertension significantly linked with the MitR244 marker on chromosome 3.

由Wistar Kyoto大鼠（WKY/lzm）和易中风自发性高血压大鼠（SHRSP/lzm）杂交产生的雄性和雌性F2后代的基础高血压（BP）与1号染色体上的白细胞唾液酸蛋白基因座紧密相关。2. 过量盐摄入导致血压升高，且雌性的反应比雄性更大。3. 在雄性F2后代中，盐敏感性高血压与10号染色体上的RR1023标记显著共分离，而不是与血管紧张素转换酶基因座共分离。4. 在雌性F2后代中，盐敏感性高血压与3号染色体上的MitR244标记显著相关。

Nara Y, Ikeda K, Matsumoto C, Nabika T, Sawamura M, Yamori Y

Graduate School of Human and Environmental Studies, Kyoto University, Japan.

Clin Exp Pharmacol Physiol. 1995 Dec;22(12):S382-5.

Basal high blood pressure (BP) in the male and female F2 progeny produced by crosses between Wistar Kyoto rats (WKY/lzm) and stroke-prone spontaneously hypertensive rats (SHRSP/lzm) linked strongly with the leukosialin locus on chromosome 1. 2. Excess salt intake caused elevation of BP and the response was larger in the females than the males. 3. In the male F2 progeny, salt-sensitive hypertension significantly cosegregated with the RR1023 marker on chromosome 10, rather than with the angiotensin converting enzyme locus. 4. In the female F2 progeny, salt-sensitive hypertension significantly linked with the MitR244 marker on chromosome 3.

由Wistar Kyoto大鼠（WKY/lzm）和易中风自发性高血压大鼠（SHRSP/lzm）杂交产生的雄性和雌性F2后代的基础高血压（BP）与1号染色体上的白细胞唾液酸蛋白基因座紧密相关。2. 过量盐摄入导致血压升高，且雌性的反应比雄性更大。3. 在雄性F2后代中，盐敏感性高血压与10号染色体上的RR1023标记显著共分离，而不是与血管紧张素转换酶基因座共分离。4. 在雌性F2后代中，盐敏感性高血压与3号染色体上的MitR244标记显著相关。