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The role of cytokines in rheumatoid arthritis. The Croonian Lecture 1995.

作者信息

Maini R N

机构信息

Kennedy Institute of Rheumatology, London.

出版信息

J R Coll Physicians Lond. 1996 Jul-Aug;30(4):344-51.

PMID:8875381
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC5401598/
Abstract
摘要
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/8595/5401598/6a59c0e4489e/jrcollphyslond90378-0068-b.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/8595/5401598/7d588bee144f/jrcollphyslond90378-0064-a.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/8595/5401598/afba925670cd/jrcollphyslond90378-0065-a.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/8595/5401598/3aa84b060738/jrcollphyslond90378-0065-b.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/8595/5401598/ec3a9de7b3f2/jrcollphyslond90378-0066-a.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/8595/5401598/f1d07812de9f/jrcollphyslond90378-0067-a.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/8595/5401598/233c8d786b0a/jrcollphyslond90378-0068-a.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/8595/5401598/6a59c0e4489e/jrcollphyslond90378-0068-b.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/8595/5401598/7d588bee144f/jrcollphyslond90378-0064-a.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/8595/5401598/afba925670cd/jrcollphyslond90378-0065-a.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/8595/5401598/3aa84b060738/jrcollphyslond90378-0065-b.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/8595/5401598/ec3a9de7b3f2/jrcollphyslond90378-0066-a.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/8595/5401598/f1d07812de9f/jrcollphyslond90378-0067-a.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/8595/5401598/233c8d786b0a/jrcollphyslond90378-0068-a.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/8595/5401598/6a59c0e4489e/jrcollphyslond90378-0068-b.jpg

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本文引用的文献

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Modulation of proinflammatory cytokine release in rheumatoid synovial membrane cell cultures. Comparison of monoclonal anti TNF-alpha antibody with the interleukin-1 receptor antagonist.类风湿性滑膜膜细胞培养中促炎细胞因子释放的调节。单克隆抗TNF-α抗体与白细胞介素-1受体拮抗剂的比较。
Eur Cytokine Netw. 1995 Jul-Dec;6(4):225-30.
2
Role of cytokines in rheumatoid arthritis.细胞因子在类风湿关节炎中的作用。
Annu Rev Immunol. 1996;14:397-440. doi: 10.1146/annurev.immunol.14.1.397.
3
Cell surface glycoproteins expressed on activated human T cells induce production of interleukin-1 beta by monocytic cells: a possible role of CD69.
类风湿关节炎患者的肿瘤坏死因子-α、白细胞介素-2可溶性受体及不同炎症参数
Mediators Inflamm. 2002 Dec;11(6):345-9. doi: 10.1080/0962935021000051539.
4
Infliximab: a review of its use in the management of rheumatoid arthritis.英夫利昔单抗:类风湿关节炎治疗应用综述
Drugs. 2000 Jun;59(6):1341-59. doi: 10.2165/00003495-200059060-00010.
5
No strong association between alleles of tumour necrosis factor alpha and interleukin-1 receptor antagonist and corneal melting associated with systemic vasculitis.肿瘤坏死因子α和白细胞介素-1受体拮抗剂的等位基因与系统性血管炎相关的角膜溶解之间无明显关联。
Br J Ophthalmol. 2000 Apr;84(4):395-8. doi: 10.1136/bjo.84.4.395.
6
The cytokine network in rheumatoid arthritis: definition of TNF alpha as a therapeutic target.类风湿关节炎中的细胞因子网络:将肿瘤坏死因子α定义为治疗靶点
J R Coll Physicians Lond. 1996 Nov-Dec;30(6):560-70.
活化的人T细胞表面表达的细胞表面糖蛋白可诱导单核细胞产生白细胞介素-1β:CD69的可能作用。
Eur Cytokine Netw. 1993 Jan-Feb;4(1):15-23.
4
Twin concordance rates for rheumatoid arthritis: results from a nationwide study.类风湿关节炎的双胞胎一致性率:一项全国性研究的结果。
Br J Rheumatol. 1993 Oct;32(10):903-7. doi: 10.1093/rheumatology/32.10.903.
5
Mice deficient for the 55 kd tumor necrosis factor receptor are resistant to endotoxic shock, yet succumb to L. monocytogenes infection.缺乏55kd肿瘤坏死因子受体的小鼠对内毒素休克具有抗性,但会死于单核细胞增生李斯特菌感染。
Cell. 1993 May 7;73(3):457-67. doi: 10.1016/0092-8674(93)90134-c.
6
Treatment of rheumatoid arthritis with chimeric monoclonal antibodies to tumor necrosis factor alpha.用抗肿瘤坏死因子α嵌合单克隆抗体治疗类风湿性关节炎。
Arthritis Rheum. 1993 Dec;36(12):1681-90. doi: 10.1002/art.1780361206.
7
Construction and initial characterization of a mouse-human chimeric anti-TNF antibody.小鼠-人嵌合抗TNF抗体的构建及初步表征
Mol Immunol. 1993 Nov;30(16):1443-53. doi: 10.1016/0161-5890(93)90106-l.
8
Immunoregulatory role of interleukin 10 in rheumatoid arthritis.白细胞介素10在类风湿性关节炎中的免疫调节作用。
J Exp Med. 1994 May 1;179(5):1517-27. doi: 10.1084/jem.179.5.1517.
9
Chronic exposure to tumor necrosis factor (TNF) in vitro impairs the activation of T cells through the T cell receptor/CD3 complex; reversal in vivo by anti-TNF antibodies in patients with rheumatoid arthritis.体外长期暴露于肿瘤坏死因子(TNF)会通过T细胞受体/CD3复合物损害T细胞的激活;类风湿性关节炎患者体内抗TNF抗体可使其逆转。
J Clin Invest. 1994 Aug;94(2):749-60. doi: 10.1172/JCI117394.
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Repeated therapy with monoclonal antibody to tumour necrosis factor alpha (cA2) in patients with rheumatoid arthritis.肿瘤坏死因子α单克隆抗体(cA2)对类风湿性关节炎患者的重复治疗。
Lancet. 1994 Oct 22;344(8930):1125-7. doi: 10.1016/s0140-6736(94)90632-7.