Signal transduction into the nucleus: fifth colloquium on cellular signal transduction. Heidelberg, Germany, January 1996.
作者信息
Marks F, Angel P
机构信息
DKFZ, Department 0235, Heidelberg, Germany.
出版信息
J Cancer Res Clin Oncol. 1996;122(10):638-42. doi: 10.1007/BF01221198.
Abstract
摘要
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Differential activation of acute phase response factor/Stat3 and Stat1 via the cytoplasmic domain of the interleukin 6 signal transducer gp130. II. Src homology SH2 domains define the specificity of stat factor activation.通过白细胞介素6信号转导子gp130的胞质结构域对急性期反应因子/信号转导和转录激活因子3（Stat3）及信号转导和转录激活因子1（Stat1）的差异性激活。II. Src同源SH2结构域决定信号转导和转录激活因子激活的特异性。
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Association of transcription factor APRF and protein kinase Jak1 with the interleukin-6 signal transducer gp130.转录因子APRF和蛋白激酶Jak1与白细胞介素-6信号转导子gp130的关联。
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Differential transactivation potential of Oct1 and Oct2 is determined by additional B cell-specific activities.Oct1和Oct2的差异反式激活潜能由其他B细胞特异性活性决定。
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Phenotypic alterations in fos-transgenic mice correlate with changes in Fos/Jun-dependent collagenase type I expression. Regulation of mouse metalloproteinases by carcinogens, tumor promoters, cAMP, and Fos oncoprotein.fos转基因小鼠的表型改变与Fos/Jun依赖性I型胶原酶表达的变化相关。致癌物、肿瘤启动子、cAMP和Fos癌蛋白对小鼠金属蛋白酶的调节。
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