Norman M, Thornton A R, Phillips A J, Slaven A
MRC Institute of Hearing Research, Royal South Hants Hospital, Southampton, UK.
Am J Otol. 1996 Sep;17(5):763-72.
Otoacoustic emission testing was carried out on 39 patients with confirmed acoustic neuromas, and satisfactory emissions were recorded from the neuroma ear of 59% of them. A comparison of the patients with and without emissions showed no significant differences in low-frequency or high-frequency hearing loss, optimum speech discrimination score, or canal paresis in the affected ear between the two groups. Emissions were recorded at stimulus rates up to 5,000 clicks/s by using the maximum length sequence (MLS) technique. The decrease in the emission amplitude with increase in click rate (rate suppression) was significantly less than the amount that would be expected from normal subjects for several of the neuroma patients. However, one patient showed normal suppression despite having a large neuroma and no measurable hearing. This would suggest that efferent suppression may not be the only mechanism involved.
对39例确诊为听神经瘤的患者进行了耳声发射测试,其中59%的患者患侧耳记录到了满意的耳声发射。有耳声发射和无耳声发射的患者之间比较发现,两组患者患耳的低频或高频听力损失、最佳言语辨别得分或外耳道轻瘫方面均无显著差异。采用最大长度序列(MLS)技术,在高达5000次/秒的刺激速率下记录耳声发射。部分听神经瘤患者耳声发射幅度随点击速率增加而降低(速率抑制)的程度明显小于正常受试者预期的降低量。然而,有一名患者尽管患有大型听神经瘤且听力无法测量,但显示出正常的抑制情况。这表明传出抑制可能不是唯一涉及的机制。