Kishi A, Nao-i N, Sawada A
Department of Ophthalmology, Miyazaki Medical College, Japan.
Am J Ophthalmol. 1996 Nov;122(5):735-7. doi: 10.1016/s0002-9394(14)70498-2.
We studied a case of Vogt-Koyanagi-Harada syndrome with transient shallow anterior chamber and increased intraocular pressure as initial manifestations.
We examined the patient using ultrasound biomicroscopy and speculated on the mechanisms of increased intraocular pressure.
Ultrasound biomicroscopic examinations revealed the swollen ciliary body with anterior rotation and supraciliary space at the time of increased intraocular pressure.
We confirmed that the swollen ciliary body with anterior rotation and supraciliary space caused transient angle-closure glaucoma in this patient with Vogt-Koyanagi-Harada syndrome.
我们研究了一例以短暂性浅前房和眼压升高为初始表现的Vogt-小柳-原田综合征病例。
我们使用超声生物显微镜对患者进行检查，并推测眼压升高的机制。
超声生物显微镜检查显示，在眼压升高时，睫状体肿胀并向前旋转，睫状体上腔增宽。
我们证实，在该例Vogt-小柳-原田综合征患者中，睫状体肿胀并向前旋转以及睫状体上腔增宽导致了短暂性闭角型青光眼。
