Pinski S L, Tchou P J, Trohman R G
Department of Cardiology, Cleveland Clinic Foundation, OH 44195, USA.
J Cardiovasc Electrophysiol. 1996 Nov;7(11):1091-4. doi: 10.1111/j.1540-8167.1996.tb00486.x.
A patient with 3:2 second-degree AV block after acute inferior wall myocardial infarction showed consistent PR interval shortening on the second conducted beat in each periodicity. Intracardiac electrophysiologic evaluation revealed that the site of block was nodal. A typical Wenckebach pattern with prolongation of the AH interval was noted. The shorter PR resulted from a paradoxical shortening of the HV interval in the second beat, most likely due to supernormal conduction in the setting of concomitant trifascicular disease.
一名急性下壁心肌梗死后出现 3:2 二度房室传导阻滞的患者，在每个心动周期的第二个下传搏动中，PR 间期持续缩短。心内电生理评估显示阻滞部位在房室结。记录到典型的文氏现象，伴有 AH 间期延长。较短的 PR 间期是由于第二个搏动中 HV 间期反常缩短所致，很可能是由于合并三分支病变情况下的超常传导。
