• 文献检索
  • 文档翻译
  • 深度研究
  • 学术资讯
  • Suppr Zotero 插件Zotero 插件
  • 邀请有礼
  • 套餐&价格
  • 历史记录
应用&插件
Suppr Zotero 插件Zotero 插件浏览器插件Mac 客户端Windows 客户端微信小程序
定价
高级版会员购买积分包购买API积分包
服务
文献检索文档翻译深度研究API 文档MCP 服务
关于我们
关于 Suppr公司介绍联系我们用户协议隐私条款
关注我们

Suppr 超能文献

核心技术专利:CN118964589B侵权必究
粤ICP备2023148730 号-1Suppr @ 2026

文献检索

告别复杂PubMed语法,用中文像聊天一样搜索,搜遍4000万医学文献。AI智能推荐,让科研检索更轻松。

立即免费搜索

文件翻译

保留排版,准确专业,支持PDF/Word/PPT等文件格式,支持 12+语言互译。

免费翻译文档

深度研究

AI帮你快速写综述,25分钟生成高质量综述,智能提取关键信息,辅助科研写作。

立即免费体验

The role of macrophages in septic shock.

作者信息

Evans T J

机构信息

Department of Infectious Diseases and Bacteriology, Royal Postgraduate Medical School, London, United Kingdom.

出版信息

Immunobiology. 1996 Oct;195(4-5):655-9. doi: 10.1016/S0171-2985(96)80029-5.

DOI:10.1016/S0171-2985(96)80029-5
PMID:8933164
Abstract
摘要

相似文献

1
The role of macrophages in septic shock.巨噬细胞在脓毒性休克中的作用。
Immunobiology. 1996 Oct;195(4-5):655-9. doi: 10.1016/S0171-2985(96)80029-5.
2
Macrophages, monokines, and the metabolic pathophysiology of septic shock.
Prog Clin Biol Res. 1989;286:203-18.
3
Ebola hemorrhagic fever and septic shock.埃博拉出血热和感染性休克。
J Infect Dis. 2003 Dec 1;188(11):1613-7. doi: 10.1086/379727. Epub 2003 Nov 14.
4
Correlation of traumatic shock with immunocompetence and sepsis.创伤性休克与免疫能力及脓毒症的相关性
Surg Forum. 1979;30:20-1.
5
[Nitric oxide--an important mediator in sepsis?].[一氧化氮——脓毒症中的重要介质?]
Tidsskr Nor Laegeforen. 1999 Nov 10;119(27):4061-5.
6
Systemic NO production during (septic) shock depends on parenchymal and not on hematopoietic cells: in vivo iNOS expression pattern in (septic) shock.(脓毒性)休克期间全身一氧化氮的产生取决于实质细胞而非造血细胞:(脓毒性)休克时体内诱导型一氧化氮合酶的表达模式。
FASEB J. 2006 Nov;20(13):2363-5. doi: 10.1096/fj.06-5798fje. Epub 2006 Oct 4.
7
Macrophages regulate the host response to bacterial lipopolysaccharides.
Prog Clin Biol Res. 1989;299:193-202.
8
[The role of platelet activating factor in regulation of the phagocytic function of macrophages from different organs].[血小板活化因子在调节不同器官巨噬细胞吞噬功能中的作用]
Biull Eksp Biol Med. 1998 Mar;125(3):315-8.
9
T cell mediated lethal shock induced by bacterial superantigens.
Behring Inst Mitt. 1992 Apr(91):46-53.
10
Bacterial DNA causes septic shock.细菌DNA会引发败血性休克。
Nature. 1997 Mar 27;386(6623):336-7. doi: 10.1038/386336a0.

引用本文的文献

1
Aging-induced dysbiosis worsens sepsis severity but is attenuated by probiotics in D-galactose-administered mice with cecal ligation and puncture model.衰老引起的肠道菌群失调会加重脓毒症的严重程度,但在盲肠结扎和穿刺模型的 D-半乳糖给药小鼠中,益生菌可以减轻这种情况。
PLoS One. 2024 Oct 18;19(10):e0311774. doi: 10.1371/journal.pone.0311774. eCollection 2024.
2
NAD prevents septic shock-induced death by non-canonical inflammasome blockade and IL-10 cytokine production in macrophages.NAD 通过非经典炎性小体阻断和巨噬细胞中白细胞介素-10 细胞因子的产生来预防脓毒性休克诱导的死亡。
Elife. 2024 Feb 19;12:RP88686. doi: 10.7554/eLife.88686.
3
Carvacrol protects mice against LPS-induced sepsis and attenuates inflammatory response in macrophages by modulating the ERK1/2 pathway.
香芹酚通过调节 ERK1/2 通路保护 LPS 诱导脓毒症的小鼠并减轻巨噬细胞的炎症反应。
Sci Rep. 2023 Aug 7;13(1):12809. doi: 10.1038/s41598-023-39665-7.
4
Sepsis Encephalopathy Is Partly Mediated by miR370-3p-Induced Mitochondrial Injury but Attenuated by BAM15 in Cecal Ligation and Puncture Sepsis Male Mice.盲肠结扎穿刺术诱导的脓毒症雄性小鼠中,miR370-3p 诱导的线粒体损伤部分介导了脓毒症脑病,但 BAM15 可减轻其损伤。
Int J Mol Sci. 2022 May 13;23(10):5445. doi: 10.3390/ijms23105445.
5
Rigosertib inhibits MEK1-ERK pathway and alleviates lipopolysaccharide-induced sepsis.里戈塞替尼抑制 MEK1-ERK 通路并缓解脂多糖诱导的败血症。
Immun Inflamm Dis. 2021 Sep;9(3):991-999. doi: 10.1002/iid3.458. Epub 2021 Jun 1.
6
VISTA Re-programs Macrophage Biology Through the Combined Regulation of Tolerance and Anti-inflammatory Pathways.VISTA通过耐受性和抗炎途径的联合调控来重新编程巨噬细胞生物学特性。
Front Immunol. 2020 Oct 15;11:580187. doi: 10.3389/fimmu.2020.580187. eCollection 2020.
7
MicroRNAs: At the Interface of Metabolic Pathways and Inflammatory Responses by Macrophages.MicroRNAs:代谢途径与巨噬细胞炎症反应的交汇点。
Front Immunol. 2020 Aug 14;11:1797. doi: 10.3389/fimmu.2020.01797. eCollection 2020.
8
Novel Role of Endothelial Derived Exosomal HSPA12B in Regulating Macrophage Inflammatory Responses in Polymicrobial Sepsis.内皮细胞衍生的外泌体 HSPA12B 在多微生物脓毒症中调节巨噬细胞炎症反应的新作用。
Front Immunol. 2020 May 7;11:825. doi: 10.3389/fimmu.2020.00825. eCollection 2020.
9
Streptococcal M protein promotes IL-10 production by cGAS-independent activation of the STING signaling pathway.链球菌 M 蛋白通过非 cGAS 依赖性激活 STING 信号通路促进 IL-10 的产生。
PLoS Pathog. 2018 Mar 26;14(3):e1006969. doi: 10.1371/journal.ppat.1006969. eCollection 2018 Mar.
10
GSTO1-1 plays a pro-inflammatory role in models of inflammation, colitis and obesity.GSTO1-1 在炎症、结肠炎和肥胖模型中发挥促炎作用。
Sci Rep. 2017 Dec 19;7(1):17832. doi: 10.1038/s41598-017-17861-6.