Saigal S, Wison R, Usher R
Radiology. 1977 Oct;125(1):185-8. doi: 10.1148/125.1.185.
As a result of excessive placental transfusion at birth, 8 premature and 3 full-term infants presented with cardiorespiratory and neurological symptoms. Cord clamping was delayed in all infants, and blood volume studies showed polycythemia or hypervolemia. The radiological findings included pulmonary vascular congestion, hyperaeration, pleural effusion, and mild cardiomegaly. Most infants responded to conservative medical management and were asymptomatic within 24 to 72 hours. Three acutely ill infants showed great improvement after phlebotomy, providing further evidence that circulatory overload due to placental transfusion may be the etiological factor in this symptom-complex.
由于出生时胎盘输血过多,8名早产儿和3名足月儿出现了心肺和神经症状。所有婴儿的脐带结扎均延迟,血容量研究显示红细胞增多症或血容量过多。影像学检查结果包括肺血管充血、过度充气、胸腔积液和轻度心脏扩大。大多数婴儿对保守治疗有反应,在24至72小时内无症状。3名重病婴儿在放血后有明显改善,这进一步证明胎盘输血导致的循环超负荷可能是这种症状复合体的病因。