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Studies on the role of protein syntheis in cell injury by toxic agents: I. Effect of cycloheximide administration on several factors modulating carbon tetrachloride-induced liver necrosis.

作者信息

Castro J A, Ferreyra E C, de Castro C R, de Fenos O M, Diaz Gómez M I, Gram T E, Reagan R L, Guarino A M

出版信息

Toxicol Appl Pharmacol. 1977 Aug;41(2):305-20. doi: 10.1016/0041-008x(77)90031-x.

DOI:10.1016/0041-008x(77)90031-x
PMID:898201
Abstract
摘要

相似文献

1
Studies on the role of protein syntheis in cell injury by toxic agents: I. Effect of cycloheximide administration on several factors modulating carbon tetrachloride-induced liver necrosis.关于蛋白质合成在毒物所致细胞损伤中的作用的研究:I. 给予环己酰亚胺对几种调节四氯化碳诱导肝坏死的因素的影响。
Toxicol Appl Pharmacol. 1977 Aug;41(2):305-20. doi: 10.1016/0041-008x(77)90031-x.
2
Effect of inhibitors of drug metabolizing enzymes on carbon tetrachloride hepatotoxicity.
Toxicol Appl Pharmacol. 1971 Mar;18(3):625-37. doi: 10.1016/s0041-008x(71)80017-0.
3
Species differences in carbon tetrachloride-induced hepatotoxicity: the role of CCl4 activation and of lipid peroxidation.四氯化碳诱导的肝毒性中的种属差异:CCl4激活及脂质过氧化的作用
Toxicol Appl Pharmacol. 1975 Oct;34(1):102-14. doi: 10.1016/0041-008x(75)90179-9.
4
Mechanism of chloramphenicol prevention of carbon tetrachloride-induced liver damage.氯霉素预防四氯化碳诱导的肝损伤的机制。
Exp Mol Pathol. 1978 Jun;28(3):395-405. doi: 10.1016/0014-4800(78)90013-8.
5
Hepatotoxicity of trichloroethylene-carbon tetrachloride mixtures in rats. A possible consequence of the potentiation by trichloroethylene of carbon tetrachloride-induced lipid peroxidation and liver lesions.三氯乙烯-四氯化碳混合物对大鼠的肝毒性。三氯乙烯增强四氯化碳诱导的脂质过氧化和肝脏损伤的一个可能后果。
Gastroenterology. 1982 Oct;83(4):761-72.
6
Carbon tetrachloride hepatotoxicity: an example of lethal cleavage.四氯化碳肝毒性:致死性裂解的一个例子。
CRC Crit Rev Toxicol. 1973 Nov;2(3):263-97. doi: 10.3109/10408447309082019.
7
Prevention of carbon tetrachloride-induced liver necrosis by the chelator alizarin sodium sulfonate.
Exp Mol Pathol. 1992 Jun;56(3):197-207. doi: 10.1016/0014-4800(92)90036-b.
8
Late preventive effects of quinacrine on carbon tetrachloride induced liver necrosis.
Arch Toxicol. 1993;67(6):386-91. doi: 10.1007/BF01977399.
9
Mechanistic studies on carbon tetrachloride hepatoxicity in fasted and fed rats.
Toxicol Appl Pharmacol. 1975 Apr;32(1):101-8. doi: 10.1016/0041-008x(75)90199-4.
10
Prevention of carbon tetrachloride-induced necrosis by inhibitors of drug metabolism--further studies on their mechanism of action.药物代谢抑制剂对四氯化碳诱导坏死的预防作用——对其作用机制的进一步研究
Biochem Pharmacol. 1974 Jan 15;23(2):295-302. doi: 10.1016/0006-2952(74)90420-1.

引用本文的文献

1
Decreased incorporation of 14C-leucine in different liver nuclear protein fractions at early stages of carbon tetrachloride poisoning in the rat.在大鼠四氯化碳中毒早期,不同肝细胞核蛋白组分中14C-亮氨酸的掺入减少。
Arch Toxicol. 1994;68(3):206-9. doi: 10.1007/s002040050056.
2
Protective effect of (+)cyanidanol-3 in acute liver injury induced by galactosamine or carbon tetrachloride in the rat.(+)-矢车菊素-3对大鼠半乳糖胺或四氯化碳诱导的急性肝损伤的保护作用。
Naunyn Schmiedebergs Arch Pharmacol. 1980 Jul;312(3):285-91. doi: 10.1007/BF00499159.
3
Protective effect of saikosaponin-d isolated from Bupleurum falcatum L. on CCl4-induced liver injury in the rat.
从柴胡中分离得到的柴胡皂苷d对四氯化碳诱导的大鼠肝损伤的保护作用。
Naunyn Schmiedebergs Arch Pharmacol. 1982 Sep;320(3):266-71. doi: 10.1007/BF00510139.