SNAREs and regulated vesicle exocytosis.
作者信息
Goda Y
机构信息
Molecular Neurobiology Laboratory, The Salk Institute, La Jolla, CA 92037, USA.
出版信息
Proc Natl Acad Sci U S A. 1997 Feb 4;94(3):769-72. doi: 10.1073/pnas.94.3.769.
Abstract
摘要
相似文献
1
SNAREs and regulated vesicle exocytosis.可溶性N-乙基马来酰胺敏感因子附着蛋白受体与调节性囊泡胞吐作用。
Proc Natl Acad Sci U S A. 1997 Feb 4;94(3):769-72. doi: 10.1073/pnas.94.3.769.
2
SNAPs and SNAREs in exocytosis in chromaffin cells.嗜铬细胞胞吐作用中的SNAPs和SNAREs
Biochem Soc Trans. 1996 Aug;24(3):653-7. doi: 10.1042/bst0240653.
3
Protein-protein interactions in synaptic vesicle exocytosis.突触小泡胞吐过程中的蛋白质-蛋白质相互作用。
Biochem Soc Trans. 1996 Aug;24(3):666-70. doi: 10.1042/bst0240666.
4
[Proteins regulating neurotransmitter release of synaptic vesicles at nerve terminals].[调节神经末梢突触小泡神经递质释放的蛋白质]
Sheng Li Ke Xue Jin Zhan. 2003 Jan;34(1):6-10.
5
Regulated exocytosis and SNARE function (Review).调节性胞吐作用与SNARE蛋白功能（综述）
Mol Membr Biol. 2003 Jul-Sep;20(3):209-20. doi: 10.1080/0968768031000104953.
6
Insight into the molecular mechanisms of synaptic vesicle docking and fusion.深入了解突触小泡对接与融合的分子机制。
Cold Spring Harb Symp Quant Biol. 1995;60:361-9. doi: 10.1101/sqb.1995.060.01.040.
7
Snares and Munc18 in synaptic vesicle fusion.突触小泡融合中的圈套蛋白和Munc18蛋白
Nat Rev Neurosci. 2002 Aug;3(8):641-53. doi: 10.1038/nrn898.
8
Regulation of neurotransmitter release kinetics by NSF.N-乙基马来酰亚胺敏感因子对神经递质释放动力学的调控
Science. 1998 Feb 20;279(5354):1203-6. doi: 10.1126/science.279.5354.1203.
9
The mechanism of the neurotransmitter release in growth cones.生长锥中神经递质释放的机制。
J Neurosci Res. 2000 Jun 15;60(6):743-53. doi: 10.1002/1097-4547(20000615)60:6<743::AID-JNR6>3.0.CO;2-T.
10
The roles of NSF, SNAPs and SNAREs during membrane fusion.N-乙基马来酰亚胺敏感因子（NSF）、可溶性NSF附着蛋白（SNAPs）和可溶性N-乙基马来酰亚胺敏感因子附着蛋白受体（SNAREs）在膜融合过程中的作用。
Biochim Biophys Acta. 1997 Jun 27;1357(2):155-72. doi: 10.1016/s0167-4889(97)00039-6.
引用本文的文献
1
Abnormal Transcytosis Mechanisms in the Pathogenesis of Hydrocephalus: A Review.脑积水发病机制中的异常转胞吞作用机制：综述
Int J Mol Sci. 2025 May 19;26(10):4881. doi: 10.3390/ijms26104881.
2
Endogenous hydrogen peroxide positively regulates secretion of a gut-derived peptide in neuroendocrine potentiation of the oxidative stress response in .内源性过氧化氢在氧化应激反应的神经内分泌增强过程中对一种肠道衍生肽的分泌起正向调节作用。
Elife. 2024 Dec 5;13:RP97503. doi: 10.7554/eLife.97503.
3
Endogenous hydrogen peroxide positively regulates secretion of a gut-derived peptide in neuroendocrine potentiation of the oxidative stress response in .内源性过氧化氢在氧化应激反应的神经内分泌增强过程中正向调节一种肠道衍生肽的分泌。
bioRxiv. 2024 Sep 22:2024.04.03.587937. doi: 10.1101/2024.04.03.587937.
4
Genetic Differences between Male and Female Pattern Hair Loss in a Korean Population.韩国人群中男性型脱发与女性型脱发的基因差异
Life (Basel). 2024 Jul 26;14(8):939. doi: 10.3390/life14080939.
5
Typhoid toxin sorting and exocytic transport from Typhi-infected cells.伤寒毒素的分拣和从伤寒感染细胞中的胞吐运输。
Elife. 2022 May 17;11:e78561. doi: 10.7554/eLife.78561.
6
Arachidonic Acid as Mechanotransducer of Renin Cell Baroreceptor.花生四烯酸作为肾素细胞压力感受器的机械转导物。
Nutrients. 2022 Feb 10;14(4):749. doi: 10.3390/nu14040749.
7
SCAMP2/5 as diagnostic and prognostic markers for acute myeloid leukemia.SCAMP2/5 作为急性髓系白血病的诊断和预后标志物。
Sci Rep. 2021 Aug 23;11(1):17012. doi: 10.1038/s41598-021-96440-2.
8
Alpha-Synuclein Post-translational Modifications: Implications for Pathogenesis of Lewy Body Disorders.α-突触核蛋白的翻译后修饰：对路易体疾病发病机制的影响
Front Aging Neurosci. 2021 Jun 25;13:690293. doi: 10.3389/fnagi.2021.690293. eCollection 2021.
9
KRAS regulation by small non-coding RNAs and SNARE proteins.KRAS 的调控作用由小非编码 RNA 和 SNARE 蛋白介导。
Nat Commun. 2019 Nov 11;10(1):5118. doi: 10.1038/s41467-019-13106-4.
10
The Roles of Post-translational Modifications on α-Synuclein in the Pathogenesis of Parkinson's Diseases.翻译后修饰对α-突触核蛋白在帕金森病发病机制中的作用
Front Neurosci. 2019 Apr 18;13:381. doi: 10.3389/fnins.2019.00381. eCollection 2019.
本文引用的文献
1
Binding of the synaptic vesicle v-SNARE, synaptotagmin, to the plasma membrane t-SNARE, SNAP-25, can explain docked vesicles at neurotoxin-treated synapses.突触小泡的v-SNARE蛋白（突触结合蛋白）与质膜的t-SNARE蛋白（SNAP-25）的结合，可以解释在经神经毒素处理的突触处停靠的小泡。
Proc Natl Acad Sci U S A. 1997 Feb 4;94(3):997-1001. doi: 10.1073/pnas.94.3.997.
2
Timing of neurotransmission at fast synapses in the mammalian brain.哺乳动物大脑中快速突触处神经传递的时间安排。
Nature. 1996 Nov 14;384(6605):170-2. doi: 10.1038/384170a0.
3
Inhibition of neurotransmission by peptides containing the synaptic protein interaction site of N-type Ca2+ channels.含N型钙离子通道突触蛋白相互作用位点的肽对神经传递的抑制作用。
Neuron. 1996 Oct;17(4):781-8. doi: 10.1016/s0896-6273(00)80209-3.
4
Functional interaction of syntaxin and SNAP-25 with voltage-sensitive L- and N-type Ca2+ channels.syntaxin和SNAP-25与电压敏感性L型和N型Ca2+通道的功能相互作用。
EMBO J. 1996 Aug 15;15(16):4100-10.
5
Synaptotagmins: C2-domain proteins that regulate membrane traffic.突触结合蛋白：调节膜转运的C2结构域蛋白。
Neuron. 1996 Sep;17(3):379-88. doi: 10.1016/s0896-6273(00)80171-3.
6
Structure and function of tetanus and botulinum neurotoxins.破伤风和肉毒杆菌神经毒素的结构与功能
Q Rev Biophys. 1995 Nov;28(4):423-72. doi: 10.1017/s0033583500003292.
7
SNAP-25 is required for a late postdocking step in Ca2+-dependent exocytosis.在钙离子依赖的胞吐作用中，SNAP-25是对接后晚期步骤所必需的。
J Biol Chem. 1996 Aug 23;271(34):20227-30. doi: 10.1074/jbc.271.34.20227.
8
N-Ethylmaleimide-sensitive factor acts at a prefusion ATP-dependent step in Ca2+-activated exocytosis.N-乙基马来酰亚胺敏感因子在钙离子激活的胞吐作用中，于融合前的ATP依赖步骤发挥作用。
J Biol Chem. 1996 Aug 23;271(34):20223-6. doi: 10.1074/jbc.271.34.20223.
9
Functional impact of syntaxin on gating of N-type and Q-type calcium channels.Syntaxin对N型和Q型钙通道门控的功能影响。
Nature. 1995 Dec 7;378(6557):623-6. doi: 10.1038/378623a0.
10
SNAP receptors implicated in vesicle targeting and fusion.参与囊泡靶向和融合的SNAP受体。
Nature. 1993 Mar 25;362(6418):318-24. doi: 10.1038/362318a0.
Zotero 插件