Src和Hck的新见解 - Suppr

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New impressions of Src and Hck.

作者信息

Pawson T

出版信息

Nature. 1997 Feb 13;385(6617):582-3, 585. doi: 10.1038/385582b0.

DOI:10.1038/385582b0

PMID:9024653

Abstract

摘要

相似文献

New impressions of Src and Hck.Src和Hck的新见解

Nature. 1997 Feb 13;385(6617):582-3, 585. doi: 10.1038/385582b0.

SH3-dependent stimulation of Src-family kinase autophosphorylation without tail release from the SH2 domain in vivo.体内SH3依赖性刺激Src家族激酶自身磷酸化，而尾部未从SH2结构域释放。

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Activation of the Src-family tyrosine kinase Hck by SH3 domain displacement.通过SH3结构域置换激活Src家族酪氨酸激酶Hck

Nature. 1997 Feb 13;385(6617):650-3. doi: 10.1038/385650a0.

Affinity of Src family kinase SH3 domains for HIV Nef in vitro does not predict kinase activation by Nef in vivo.Src家族激酶SH3结构域在体外对HIV Nef的亲和力并不能预测其在体内被Nef激活激酶的情况。

Biochemistry. 2000 Jan 25;39(3):489-95. doi: 10.1021/bi992504j.

The human immunodeficiency virus type 1 Nef protein binds the Src-related tyrosine kinase Lck SH2 domain through a novel phosphotyrosine independent mechanism.1型人类免疫缺陷病毒Nef蛋白通过一种新的非磷酸酪氨酸依赖机制与Src相关酪氨酸激酶Lck的SH2结构域结合。

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Oligomerization is required for HIV-1 Nef-induced activation of the Src family protein-tyrosine kinase, Hck.HIV-1 Nef诱导Src家族蛋白酪氨酸激酶Hck激活需要寡聚化。

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SH3 domains with high affinity and engineered ligand specificities targeted to HIV-1 Nef.具有高亲和力且针对HIV-1 Nef设计了配体特异性的SH3结构域。

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RT loop flexibility enhances the specificity of Src family SH3 domains for HIV-1 Nef.RT环的灵活性增强了Src家族SH3结构域对HIV-1 Nef的特异性。

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Crystal structure of the Src family tyrosine kinase Hck.Src家族酪氨酸激酶Hck的晶体结构

Nature. 1997 Feb 13;385(6617):602-9. doi: 10.1038/385602a0.

Reading between the lines: SH3 recognition of an intact protein.字里行间的解读：SH3对完整蛋白质的识别

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Knockdown of GnT-Va expression inhibits ligand-induced downregulation of the epidermal growth factor receptor and intracellular signaling by inhibiting receptor endocytosis.敲低GnT-Va的表达可通过抑制受体的内吞作用来抑制配体诱导的表皮生长因子受体下调和细胞内信号传导。

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Process outgrowth of oligodendrocytes is promoted by interaction of fyn kinase with the cytoskeletal protein tau.Fyn激酶与细胞骨架蛋白tau相互作用可促进少突胶质细胞的突起生长。

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