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1
Accumulation of glucosaminyl(acyl)phosphatidylinositol in an S3 HeLa subline expressing normal dolicholphosphomannose synthase activity.在表达正常多萜醇磷酸甘露糖合酶活性的S3 HeLa亚系中糖胺基(酰基)磷脂酰肌醇的积累。
Biochem J. 1997 Feb 1;321 ( Pt 3)(Pt 3):837-44. doi: 10.1042/bj3210837.
2
Uptake of exogenous sn-1-acyl-2-lyso-phosphatidylinositol into HeLa S3 cells. Reacylation on the cell surface and metabolism to glucosaminyl(acyl)phosphatidylinositol.外源性sn-1-酰基-2-溶血磷脂酰肌醇被HeLa S3细胞摄取。在细胞表面进行再酰化并代谢为葡糖胺基(酰基)磷脂酰肌醇。
J Biol Chem. 1995 Apr 21;270(16):9147-53. doi: 10.1074/jbc.270.16.9147.
3
Metabolism of exogenous sn-1-alkyl-sn-2-lyso-glucosaminyl-phosphatidylinositol in HeLa D cells: accumulation of glucosaminyl(acyl)phosphatidylinositol in a metabolically inert compartment.外源sn-1-烷基-sn-2-溶血氨基葡糖基磷脂酰肌醇在HeLa D细胞中的代谢:氨基葡糖基(酰基)磷脂酰肌醇在代谢惰性区室中的积累
Biochem J. 2001 Oct 15;359(Pt 2):305-13. doi: 10.1042/0264-6021:3590305.
4
Compositional analysis of glucosaminyl(acyl)phosphatidylinositol accumulated in HeLa S3 cells.
Eur J Biochem. 1995 Oct 1;233(1):384-94. doi: 10.1111/j.1432-1033.1995.384_1.x.
5
Inositol acylation of a potential glycosyl phosphoinositol anchor precursor from yeast requires acyl coenzyme A.来自酵母的一种潜在糖基磷脂酰肌醇锚定前体的肌醇酰化作用需要酰基辅酶A。
J Biol Chem. 1992 Apr 25;267(12):8599-603.
6
Glycosylphosphatidylinositol-anchor intermediates associate with triton-insoluble membranes in subcellular compartments that include the endoplasmic reticulum.糖基磷脂酰肌醇锚定中间体与包括内质网在内的亚细胞区室中的不溶于曲拉通的膜相关联。
Biochem J. 1999 Nov 1;343 Pt 3(Pt 3):627-35.
7
Acylation of glucosaminyl phosphatidylinositol revisited. Palmitoyl-CoA dependent palmitoylation of the inositol residue of a synthetic dioctanoyl glucosaminyl phosphatidylinositol by hamster membranes permits efficient mannosylation of the glucosamine residue.重新审视葡糖胺基磷脂酰肌醇的酰化作用。仓鼠细胞膜可使合成的二辛酰葡糖胺基磷脂酰肌醇的肌醇残基发生依赖棕榈酰辅酶A的棕榈酰化,从而使葡糖胺残基高效甘露糖基化。
J Biol Chem. 1996 Oct 25;271(43):27031-8. doi: 10.1074/jbc.271.43.27031.
8
Crystal structure of phosphatidylinositol-specific phospholipase C from Bacillus cereus in complex with glucosaminyl(alpha 1-->6)-D-myo-inositol, an essential fragment of GPI anchors.蜡样芽孢杆菌磷脂酰肌醇特异性磷脂酶C与GPI锚定物的必需片段氨基葡萄糖基(α1→6)-D-肌醇复合物的晶体结构
Biochemistry. 1996 Jul 23;35(29):9496-504. doi: 10.1021/bi9606105.
9
GPI biosynthesis in mammalian cells: CoA-dependent acylation of GlcN-PI.哺乳动物细胞中的糖基磷脂酰肌醇生物合成:GlcN-PI的辅酶A依赖性酰化作用
Braz J Med Biol Res. 1994 Feb;27(2):161-6.
10
Role of exogenous inositol and phosphatidylinositol in glycosylphosphatidylinositol anchor synthesis of GP49 by Giardia lamblia.外源性肌醇和磷脂酰肌醇在蓝氏贾第鞭毛虫GP49糖基磷脂酰肌醇锚合成中的作用。
Biochim Biophys Acta. 2000 Jan 3;1483(1):69-80. doi: 10.1016/s1388-1981(99)00171-7.

引用本文的文献

1
Effect of glycosylphosphatidylinositol (GPI)-phospholipase D overexpression on GPI metabolism.糖基磷脂酰肌醇(GPI)-磷脂酶D过表达对GPI代谢的影响。
Biochem J. 2004 Mar 1;378(Pt 2):641-8. doi: 10.1042/BJ20031326.
2
Metabolism of exogenous sn-1-alkyl-sn-2-lyso-glucosaminyl-phosphatidylinositol in HeLa D cells: accumulation of glucosaminyl(acyl)phosphatidylinositol in a metabolically inert compartment.外源sn-1-烷基-sn-2-溶血氨基葡糖基磷脂酰肌醇在HeLa D细胞中的代谢:氨基葡糖基(酰基)磷脂酰肌醇在代谢惰性区室中的积累
Biochem J. 2001 Oct 15;359(Pt 2):305-13. doi: 10.1042/0264-6021:3590305.
3
Glycosylphosphatidylinositol-anchor intermediates associate with triton-insoluble membranes in subcellular compartments that include the endoplasmic reticulum.糖基磷脂酰肌醇锚定中间体与包括内质网在内的亚细胞区室中的不溶于曲拉通的膜相关联。
Biochem J. 1999 Nov 1;343 Pt 3(Pt 3):627-35.

本文引用的文献

1
A novel class of cell surface glycolipids of mammalian cells. Free glycosyl phosphatidylinositols.一类新型的哺乳动物细胞表面糖脂。游离糖基磷脂酰肌醇。
J Biol Chem. 1996 May 31;271(22):12879-84. doi: 10.1074/jbc.271.22.12879.
2
Mannosamine inhibits the synthesis of putative glycoinositol phospholipid anchor precursors in mammalian cells without incorporating into an accumulated intermediate.甘露糖胺抑制哺乳动物细胞中假定的糖基磷脂酰肌醇锚定前体的合成,且不会掺入积累的中间体中。
J Biol Chem. 1993 May 25;268(15):10938-45.
3
Defective mannosylation of glycosylphosphatidylinositol in Lec35 Chinese hamster ovary cells.Lec35中国仓鼠卵巢细胞中糖基磷脂酰肌醇的甘露糖基化缺陷。
J Biol Chem. 1993 Mar 25;268(9):6721-8.
4
Palmitylation of an amino-terminal cysteine motif of protein tyrosine kinases p56lck and p59fyn mediates interaction with glycosyl-phosphatidylinositol-anchored proteins.蛋白酪氨酸激酶p56lck和p59fyn氨基末端半胱氨酸基序的棕榈酰化介导了与糖基磷脂酰肌醇锚定蛋白的相互作用。
Mol Cell Biol. 1993 Oct;13(10):6385-92. doi: 10.1128/mcb.13.10.6385-6392.1993.
5
The structure, biosynthesis and function of glycosylated phosphatidylinositols in the parasitic protozoa and higher eukaryotes.寄生原生动物和高等真核生物中糖基化磷脂酰肌醇的结构、生物合成及功能
Biochem J. 1993 Sep 1;294 ( Pt 2)(Pt 2):305-24. doi: 10.1042/bj2940305.
6
Metabolism of GPIs in mammalian cells.哺乳动物细胞中糖基磷脂酰肌醇(GPIs)的代谢。
Braz J Med Biol Res. 1994 Feb;27(2):151-9.
7
The GPI anchor of cell-surface proteins is synthesized on the cytoplasmic face of the endoplasmic reticulum.细胞表面蛋白的糖基磷脂酰肌醇(GPI)锚定物在内质网的胞质面合成。
J Cell Biol. 1994 Oct;127(2):333-41. doi: 10.1083/jcb.127.2.333.
8
Glycoinositol phospholipid anchor-defective K562 mutants with biochemical lesions distinct from those in Thy-1- murine lymphoma mutants.糖基肌醇磷脂锚定缺陷型K562突变体,其生化损伤与Thy-1-小鼠淋巴瘤突变体不同。
J Biol Chem. 1994 Mar 4;269(9):6536-42.
9
Uptake of exogenous sn-1-acyl-2-lyso-phosphatidylinositol into HeLa S3 cells. Reacylation on the cell surface and metabolism to glucosaminyl(acyl)phosphatidylinositol.外源性sn-1-酰基-2-溶血磷脂酰肌醇被HeLa S3细胞摄取。在细胞表面进行再酰化并代谢为葡糖胺基(酰基)磷脂酰肌醇。
J Biol Chem. 1995 Apr 21;270(16):9147-53. doi: 10.1074/jbc.270.16.9147.
10
A candidate mammalian glycoinositol phospholipid precursor containing three phosphoethanolamines.一种含有三个磷酸乙醇胺的潜在哺乳动物糖基磷脂酰肌醇前体。
J Biol Chem. 1993 May 15;268(14):9998-10002.

在表达正常多萜醇磷酸甘露糖合酶活性的S3 HeLa亚系中糖胺基(酰基)磷脂酰肌醇的积累。

Accumulation of glucosaminyl(acyl)phosphatidylinositol in an S3 HeLa subline expressing normal dolicholphosphomannose synthase activity.

作者信息

Sevlever D, Schiemann D, Guidubaldi J, Medof M E, Rosenberry T L

机构信息

Department of Pharmacology, Case Western Reserve University, Cleveland, OH 44106, U.S.A.

出版信息

Biochem J. 1997 Feb 1;321 ( Pt 3)(Pt 3):837-44. doi: 10.1042/bj3210837.

DOI:10.1042/bj3210837
PMID:9032473
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC1218142/
Abstract

Glucosaminyl(acyl)phosphatidylinositol [GlcN(acyl)PI], the third intermediate in the mammalian glycosylphosphatidylinositol (GPI) anchor pathway, is undetectable in most cells. This intermediate was previously shown to accumulate, however, in murine lymphoma mutant E and in yeast mutant dpm1, both of which lack dolicholphosphomannose synthase activity. Here we report that a mammalian HeLa S3 subline, denoted D, produces large amounts of GlcN(acyl)PI. The level of GlcN(acyl)PI in this subline is twice that in the murine lymphoma mutant E and 4 times that in the parental S3 line. This HeLa D subline differs from the previously reported mutants that accumulate GlcN(acyl)PI because no defects in the synthesis or utilization of dolicholphosphomannose were found. Kinetic analysis indicated that in this HeLa subline there is an increased rate of synthesis of GlcN(acyl)PI, whereas the rate of metabolism for this GPI is comparable to that in wild-type cells. Furthermore, HeLa D cells accumulate GlcN(acyl)PI without a block in the synthesis of the downstream mannosylated GPI anchor precursors and GPI-anchored proteins. These findings might be relevant for understanding the regulation of the GPI pathway.

摘要

N-乙酰葡糖胺(酰基)磷脂酰肌醇[GlcN(acyl)PI]是哺乳动物糖基磷脂酰肌醇(GPI)锚定途径中的第三个中间体,在大多数细胞中无法检测到。然而,先前已证明该中间体在小鼠淋巴瘤突变体E和酵母突变体dpm1中积累,这两种突变体均缺乏多萜醇磷酸甘露糖合酶活性。在此我们报告,一种名为D的哺乳动物HeLa S3亚系会产生大量的GlcN(acyl)PI。该亚系中GlcN(acyl)PI的水平是小鼠淋巴瘤突变体E中的两倍,是亲本S3系中的四倍。这个HeLa D亚系与先前报道的积累GlcN(acyl)PI的突变体不同,因为未发现多萜醇磷酸甘露糖的合成或利用存在缺陷。动力学分析表明,在这个HeLa亚系中,GlcN(acyl)PI的合成速率增加,而这种GPI的代谢速率与野生型细胞中的相当。此外,HeLa D细胞积累GlcN(acyl)PI时,下游甘露糖基化GPI锚定前体和GPI锚定蛋白的合成没有受阻。这些发现可能与理解GPI途径的调控有关。