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Pathophysiology of the intermediate syndrome of organophosphorus poisoning.

作者信息

Sedgwick E M, Senanayake N

出版信息

J Neurol Neurosurg Psychiatry. 1997 Feb;62(2):201-2. doi: 10.1136/jnnp.62.2.201.

DOI:10.1136/jnnp.62.2.201
PMID:9048731
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC486742/
Abstract
摘要

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本文引用的文献

1
Independent regulation of activation and inactivation phases in non-contractile Ca2+ transients by nicotinic receptor at the mouse neuromuscular junction.小鼠神经肌肉接头处烟碱受体对非收缩性Ca2+瞬变中激活和失活阶段的独立调节。
Brain Res. 1994 Jul 11;650(2):299-304. doi: 10.1016/0006-8993(94)91795-7.
2
Acetylcholine receptor gene expression at the developing neuromuscular junction.发育中的神经肌肉接头处乙酰胆碱受体基因的表达
Physiol Rev. 1995 Apr;75(2):339-68. doi: 10.1152/physrev.1995.75.2.339.
3
Neurotoxic effects of organophosphorus insecticides. An intermediate syndrome.有机磷杀虫剂的神经毒性作用。中间综合征。
N Engl J Med. 1987 Mar 26;316(13):761-3. doi: 10.1056/NEJM198703263161301.
4
Up-and-down regulation of skeletal muscle acetylcholine receptors. Effects on neuromuscular blockers.骨骼肌乙酰胆碱受体的上调和下调。对神经肌肉阻滞剂的影响。
Anesthesiology. 1992 May;76(5):822-43. doi: 10.1097/00000542-199205000-00022.