相似文献
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Evolutionary conservation of minor U12-type spliceosome between plants and humans.植物与人类之间小U12型剪接体的进化保守性。
RNA. 2005 Jul;11(7):1095-107. doi: 10.1261/rna.2440305.
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Conserved sequences in a class of rare eukaryotic nuclear introns with non-consensus splice sites.一类具有非共有剪接位点的罕见真核细胞核内含子中的保守序列。
J Mol Biol. 1994 Jun 10;239(3):357-65. doi: 10.1006/jmbi.1994.1377.
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An ancient mechanism for splicing control: U11 snRNP as an activator of alternative splicing.一种古老的剪接调控机制：U11 snRNP 作为可变剪接的激活因子。
Mol Cell. 2010 Mar 26;37(6):821-33. doi: 10.1016/j.molcel.2010.02.014.
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The U11/U12 snRNP 65K protein acts as a molecular bridge, binding the U12 snRNA and U11-59K protein.U11/U12小核核糖核蛋白65K蛋白充当分子桥梁，结合U12小核RNA和U11 - 59K蛋白。
EMBO J. 2005 Sep 7;24(17):3057-69. doi: 10.1038/sj.emboj.7600765. Epub 2005 Aug 11.
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The U1 snRNP protein U1C recognizes the 5' splice site in the absence of base pairing.U1 snRNP蛋白U1C在不存在碱基配对的情况下识别5'剪接位点。
Nature. 2002 Sep 5;419(6902):86-90. doi: 10.1038/nature00947.
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Base pairing with U6atac snRNA is required for 5' splice site activation of U12-dependent introns in vivo.在体内，与U6atac snRNA的碱基配对是U12依赖型内含子5'剪接位点激活所必需的。
RNA. 1998 Jun;4(6):709-18. doi: 10.1017/s1355838298980207.
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Base pairing between U2 and U6 snRNAs is necessary for splicing of a mammalian pre-mRNA.U2与U6小核RNA之间的碱基配对对于哺乳动物前体信使核糖核酸的剪接是必需的。
Nature. 1991 Aug 29;352(6338):818-21. doi: 10.1038/352818a0.
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Evidence for an essential non-Watson-Crick interaction between the first and last nucleotides of a nuclear pre-mRNA intron.核前体mRNA内含子第一个和最后一个核苷酸之间存在关键的非沃森-克里克相互作用的证据。
Nature. 1993 Feb 18;361(6413):660-2. doi: 10.1038/361660a0.
引用本文的文献
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Taxonomy of introns and the evolution of minor introns.内含子的分类和小内含子的演化。
Nucleic Acids Res. 2024 Aug 27;52(15):9247-9266. doi: 10.1093/nar/gkae550.
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Distinct functions for the paralogous RBM41 and U11/U12-65K proteins in the minor spliceosome.旁系同源蛋白RBM41和U11/U12-65K在次要剪接体中的不同功能。
Nucleic Acids Res. 2024 Apr 24;52(7):4037-4052. doi: 10.1093/nar/gkae070.
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Introns: the "dark matter" of the eukaryotic genome.内含子：真核生物基因组的“暗物质”。
Front Genet. 2023 May 16;14:1150212. doi: 10.3389/fgene.2023.1150212. eCollection 2023.
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SSRTool: A web tool for evaluating RNA secondary structure predictions based on species-specific functional interpretability.SSRTool：一种基于物种特异性功能可解释性评估RNA二级结构预测的网络工具。
Comput Struct Biotechnol J. 2022 May 18;20:2473-2483. doi: 10.1016/j.csbj.2022.05.028. eCollection 2022.
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The integrity of the U12 snRNA 3' stem-loop is necessary for its overall stability.U12 snRNA 3' 茎环结构的完整性对于其整体稳定性是必要的。
Nucleic Acids Res. 2021 Mar 18;49(5):2835-2847. doi: 10.1093/nar/gkab048.
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Defective minor spliceosomes induce SMA-associated phenotypes through sensitive intron-containing neural genes in Drosophila.缺陷性小剪接体通过果蝇中敏感的内含子神经基因诱导 SMA 相关表型。
Nat Commun. 2020 Nov 5;11(1):5608. doi: 10.1038/s41467-020-19451-z.
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Minor intron splicing revisited: identification of new minor intron-containing genes and tissue-dependent retention and alternative splicing of minor introns.重新审视次要内含子剪接：新的包含次要内含子的基因的鉴定以及次要内含子的组织依赖性保留和可变剪接。
BMC Genomics. 2019 Aug 30;20(1):686. doi: 10.1186/s12864-019-6046-x.
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Minor spliceosome inactivation causes microcephaly, owing to cell cycle defects and death of self-amplifying radial glial cells.小剪接体失活导致小头畸形，这是由于细胞周期缺陷和自我扩增的放射状胶质细胞死亡所致。
Development. 2018 Aug 28;145(17):dev166322. doi: 10.1242/dev.166322.
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Mutations in the U11/U12-65K protein associated with isolated growth hormone deficiency lead to structural destabilization and impaired binding of U12 snRNA.与孤立性生长激素缺乏症相关的 U11/U12-65K 蛋白突变导致结构不稳定和 U12 snRNA 结合受损。
RNA. 2018 Mar;24(3):396-409. doi: 10.1261/rna.062844.117. Epub 2017 Dec 18.
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Human ribosomal protein eS1 is engaged in cellular events related to processing and functioning of U11 snRNA.人类核糖体蛋白eS1参与了与U11小核RNA加工和功能相关的细胞活动。
Nucleic Acids Res. 2017 Sep 6;45(15):9121-9137. doi: 10.1093/nar/gkx559.
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