[胆汁运动障碍在消化性溃疡黏膜屏障保护特性损伤机制中的作用]
[The role of biliary dyskinesia in the mechanism of the damage to the protective properties of the mucosal barrier in peptic ulcer].
作者信息
Oparin A G, Demerchian T I, Korenovskiĭ I P, Chonka V Iu, Iakovenko E L, Pandeĭ I
出版信息
Ter Arkh. 1993;65(2):44-6.
Mucous barrier resistance was studied in 232 duodenal ulcer patients with signs of exacerbation as shown by PAS-positive substances and acid mucopolysaccharides in antral mucosa of the stomach and duodenum as well as by gastric juice levels of gastromucoproteins, fucose and sialic acids. In addition, measurements were performed of biliary motility. The tests indicated that half of the ulcer patients developed biliary dyskinesia manifesting as hyperkinesia or hypokinesia. The resultant decrease in gastric mucins requires individual correction.
通过胃窦黏膜和十二指肠中PAS阳性物质、酸性黏多糖以及胃液中胃黏蛋白、岩藻糖和唾液酸水平，对232例有病情加重迹象的十二指肠溃疡患者的黏液屏障阻力进行了研究。此外，还进行了胆汁运动性测量。测试表明，一半的溃疡患者出现胆汁运动障碍，表现为运动亢进或运动减退。由此导致的胃黏液素减少需要进行个体化纠正。
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