Understanding the genetic basis of mood disorders: where do we stand?
作者信息
Reus V I, Freimer N B
机构信息
Department of Psychiatry, University of California, San Francisco 94143-0984, USA.
出版信息
Am J Hum Genet. 1997 Jun;60(6):1283-8. doi: 10.1086/515482.
Abstract
摘要
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Understanding the genetic basis of mood disorders: where do we stand?了解情绪障碍的遗传基础：我们目前的进展如何？
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本文引用的文献
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Behavioral genetics '97: ASHG statement. Recent developments in human behavioral genetics: past accomplishments and future directions.1997年行为遗传学：美国人类遗传学会声明。人类行为遗传学的最新进展：过去的成就与未来的方向。
Am J Hum Genet. 1997 Jun;60(6):1265-75. doi: 10.1086/515473.
2
Report of the Fourth International Workshop on Human Chromosome 18 Mapping. Boston, Massachusetts, October 7-9, 1996.第四届人类18号染色体图谱绘制国际研讨会报告。马萨诸塞州波士顿，1996年10月7日至9日。
Cytogenet Cell Genet. 1996;75(2-3):111-31. doi: 10.1159/000134462.
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Possible locus for bipolar disorder near the dopamine transporter on chromosome 5.5号染色体上多巴胺转运体附近可能存在双相情感障碍的基因座。
Am J Med Genet. 1996 Nov 22;67(6):533-40. doi: 10.1002/(SICI)1096-8628(19961122)67:6<533::AID-AJMG4>3.0.CO;2-I.
4
Unipolar relatives in bipolar pedigrees: a search for elusive indicators of underlying bipolarity.双相谱系中的单相亲属：寻找潜在双相性的难以捉摸的指标。
Am J Med Genet. 1996 Sep 20;67(5):445-54. doi: 10.1002/(SICI)1096-8628(19960920)67:5<445::AID-AJMG2>3.0.CO;2-J.
5
Attitudes towards bipolar disorder and predictive genetic testing among patients and providers.患者与医疗服务提供者对双相情感障碍及预测性基因检测的态度。
J Med Genet. 1996 Jul;33(7):544-9. doi: 10.1136/jmg.33.7.544.
6
A putative vulnerability locus to multiple sclerosis maps to 5p14-p12 in a region syntenic to the murine locus Eae2.一个假定的多发性硬化症易感基因座定位于5p14 - p12，该区域与小鼠基因座Eae2同线。
Nat Genet. 1996 Aug;13(4):477-80. doi: 10.1038/ng0896-477.
7
A full genome search in multiple sclerosis.多发性硬化症的全基因组搜索。
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A complete genomic screen for multiple sclerosis underscores a role for the major histocompatability complex. The Multiple Sclerosis Genetics Group.一项针对多发性硬化症的全基因组筛查突出了主要组织相容性复合体的作用。多发性硬化症遗传学小组。
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