Olsen F
Acta Pathol Microbiol Scand A. 1977 Sep;85(5):572-6. doi: 10.1111/j.1699-0463.1977.tb00448.x.
Experimentally induced acute angiotensin hypertension has been shown to increase the permeability of cerebral arterioles, venules and veins to plasma components whthin a few hours. This increase in permiability was demonstrated by means of circulating homologous fluorescent serum proteins and colloidal carbon particles. The results support the view that an increased permeability of the cerebral vessels to plasma components is either a causal or an additional pathogenetic factor in the development of the hypertensive encephalopathy.
实验性诱导的急性血管紧张素性高血压已被证明在数小时内会增加脑小动脉、小静脉和静脉对血浆成分的通透性。这种通透性的增加通过循环同源荧光血清蛋白和胶体碳颗粒得以证实。这些结果支持这样一种观点，即脑血管对血浆成分通透性的增加在高血压脑病的发生发展中要么是一个致病因素，要么是一个附加的致病因素。
