Gjörstrup P
Acta Physiol Scand. 1977 Oct;101(2):211-8. doi: 10.1111/j.1748-1716.1977.tb06000.x.
Sympathetic secretory responses from the parotid and the submaxillary glands of the rabbit were studied; sympathetic excitation occurred either alone or during a slow parasympathetic background secretion, imitating the resting secretion normally present. The fairly rapid sympathetically evoked flow of saliva from the parotid gland was, in the background secretion, further increased and obtained at low frequencies, which at sympathetic stimulation alone were subthreshold. The effects, which could be repeated with sympathomimetic drugs in a pilocarpine induced secretion, were abolished by alpha and beta-adrenoceptor blockade in combination. The submaxillary gland gave a very scanty secretion on sympathetic excitation, the effects being mediated via beta-adrenoceptors. The responses were not increased in a background secretion but the main effect of sympathetic activation was to retard the salivary flow. The retardation was attributed to vasoconstriction accompanying sympathetic excitation. Eliminating the vasoconstrictor responses, while retaining the sympathetic secretory effects, did not reveal any increase of the sympathetic salivary responses in the submaxillary gland.
对家兔腮腺和颌下腺的交感分泌反应进行了研究；交感神经兴奋可单独发生，也可在副交感神经缓慢的基础分泌过程中发生，模拟正常存在的静息分泌。在基础分泌状态下，交感神经诱发腮腺相当快速的唾液分泌量在低频时进一步增加，而单独交感神经刺激时这些低频是阈下的。在毛果芸香碱诱导的分泌中，拟交感神经药物可重复这些效应，联合使用α和β肾上腺素能受体阻滞剂可消除这些效应。颌下腺在交感神经兴奋时分泌极少，其效应通过β肾上腺素能受体介导。在基础分泌状态下，反应并未增强，但交感神经激活的主要作用是减缓唾液分泌。这种减缓归因于交感神经兴奋时伴随的血管收缩。消除血管收缩反应，同时保留交感神经分泌效应，并未显示颌下腺交感神经唾液分泌反应有任何增加。
