Molecular mechanisms involved in T-B interactions.
作者信息
Lane P
机构信息
Basel Institute for Immunology, Switzerland.
出版信息
Chem Immunol. 1997;67:1-13. doi: 10.1159/000058674.
PMID:9212494
Abstract
摘要
相似文献
1
Molecular mechanisms involved in T-B interactions.T细胞与B细胞相互作用所涉及的分子机制。
Chem Immunol. 1997;67:1-13. doi: 10.1159/000058674.
2
Role of the CD28-B7 costimulatory pathways in T cell-dependent B cell responses.CD28 - B7共刺激通路在T细胞依赖性B细胞应答中的作用。
Adv Immunol. 1996;62:131-66. doi: 10.1016/s0065-2776(08)60429-0.
3
Costimulation of T lymphocytes: the role of CD28, CTLA-4, and B7/BB1 in interleukin-2 production and immunotherapy.
Cell. 1992 Dec 24;71(7):1065-8. doi: 10.1016/s0092-8674(05)80055-8.
4
CTLA-4 is a second receptor for the B cell activation antigen B7.细胞毒性T淋巴细胞相关抗原4是B细胞活化抗原B7的第二种受体。
J Exp Med. 1991 Sep 1;174(3):561-9. doi: 10.1084/jem.174.3.561.
5
Is CTLA-4 a negative regulator for T-cell activation?细胞毒性T淋巴细胞相关抗原4（CTLA-4）是T细胞活化的负调节因子吗？
Immunol Today. 1997 Dec;18(12):569-72. doi: 10.1016/s0167-5699(97)01170-5.
6
The CD28/CTLA-4:B7 receptor system in experimental autoimmune encephalomyelitis.实验性自身免疫性脑脊髓炎中的CD28/CTLA-4:B7受体系统
J Clin Invest. 1995 Jun;95(6):2429-30. doi: 10.1172/JCI117940.
7
More negative feedback?更多负面反馈？
Nat Immunol. 2003 Jul;4(7):647-8. doi: 10.1038/ni0703-647.
8
The emerging role of CTLA-4 as an immune attenuator.细胞毒性T淋巴细胞相关抗原4（CTLA-4）作为一种免疫衰减因子的新作用。
Immunity. 1997 Oct;7(4):445-50. doi: 10.1016/s1074-7613(00)80366-0.
9
Lymphocyte activation: T-cell regulation by CTLA-4.淋巴细胞活化：CTLA-4对T细胞的调节
Curr Biol. 1996 Apr 1;6(4):398-400. doi: 10.1016/s0960-9822(02)00506-7.
10
New protein steals the show as 'costimulator' of T cells.新型蛋白质作为T细胞的“共刺激分子”成为焦点。
Science. 1993 Nov 5;262(5135):844-5. doi: 10.1126/science.7694360.
引用本文的文献
1
Growth and survival of B-chronic lymphocytic leukaemia cells.B 细胞慢性淋巴细胞白血病细胞的生长与存活
Med Oncol. 1998 Jul;15(2):73-8. doi: 10.1007/BF02989583.
Zotero 插件