Sato I, Matsusaka N, Tsuda S, Kobayashi H, Nishimura Y
Faculty of Agriculture, Iwate University, Morioka, Japan.
Radiat Res. 1997 Jul;148(1):98-100.
The biological half-life of 137Cs and its organ distribution were investigated in mice fed various potassium-deficient diets. The biological half-life, which was 6.1 days in mice receiving the normal level of potassium, became longer as the dietary potassium content decreased, and 137Cs was hardly excreted from the body when dietary potassium content was restricted to 200 mg/kg or less. The muscle showed the highest concentration of 137Cs in both mice that had sufficient amounts of potassium and those that were potassium-deficient. Clearance of 137Cs from tissues was generally suppressed when mice were fed a potassium-deficient diet, but the relative distribution pattern of 137Cs was not affected by dietary potassium content. These results suggest that dietary potassium intake, which may vary with eating habits, affects the biological half-life of 137Cs in humans.
在喂食不同低钾饮食的小鼠中研究了137Cs的生物半衰期及其在器官中的分布。在摄入正常水平钾的小鼠中，生物半衰期为6.1天，随着饮食中钾含量的降低而变长，当饮食中钾含量限制在200mg/kg或更低时，137Cs几乎不从体内排出。在钾含量充足和低钾的小鼠中，肌肉中137Cs的浓度最高。当给小鼠喂食低钾饮食时，组织中137Cs的清除通常受到抑制，但137Cs的相对分布模式不受饮食钾含量的影响。这些结果表明，饮食中钾的摄入量可能因饮食习惯而异，会影响人体中137Cs的生物半衰期。
