典型的着色性干皮病 A 互补组成纤维细胞具有可检测到的紫外线诱导的非程序性 DNA 合成。 - Suppr

Suppr

超能文献

Typical xeroderma pigmentosum complementation group A fibroblasts have detectable ultraviolet light-induced unscheduled DNA synthesis.

作者信息

Petinga R A, Andrews A D, Tarone R E, Robbins J H

出版信息

Biochim Biophys Acta. 1977 Dec 14;479(4):400-10. doi: 10.1016/0005-2787(77)90033-8.

DOI:10.1016/0005-2787(77)90033-8

PMID:922009

Abstract

摘要

相似文献

Typical xeroderma pigmentosum complementation group A fibroblasts have detectable ultraviolet light-induced unscheduled DNA synthesis.典型的着色性干皮病 A 互补组成纤维细胞具有可检测到的紫外线诱导的非程序性 DNA 合成。

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Mechanisms of inhibition of DNA replication by ultraviolet light in normal human and xeroderma pigmentosum fibroblasts.紫外线对正常人及着色性干皮病成纤维细胞DNA复制的抑制机制

J Mol Biol. 1981 Jun 25;149(2):171-87. doi: 10.1016/0022-2836(81)90297-7.

DNA chain elongation and joining in normal human and xeroderma pigmentosum cells after ultraviolet irradiation.紫外线照射后正常人细胞和着色性干皮病细胞中的DNA链延伸与连接

Biophys J. 1972 Sep;12(9):1183-91. doi: 10.1016/S0006-3495(72)86154-X.

DNA excision repair in human cells treated with ultraviolet radiation and 7,12-dimethylbenz[a]anthracene 5,6-oxide.用紫外线和7,12-二甲基苯并[a]蒽5,6-氧化物处理的人类细胞中的DNA切除修复

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Lethality and the induction and repair of DNA damage in far, mid or near UV-irradiated human fibroblasts: comparison of effects in normal, xeroderma pigmentosum and Bloom's syndrome cells.

Photochem Photobiol. 1982 Sep;36(3):333-43. doi: 10.1111/j.1751-1097.1982.tb04383.x.

Xeroderma pigmentosum complementation group F: more assignments and repair characteristics.着色性干皮病互补组F：更多任务及修复特征

Photochem Photobiol. 1985 May;41(5):629-34. doi: 10.1111/j.1751-1097.1985.tb03538.x.

Unscheduled synthesis of DNA and poly(ADP-ribose) in human fibroblasts following DNA damage.DNA损伤后人成纤维细胞中DNA和聚（ADP - 核糖）的非程序性合成。

J Supramol Struct Cell Biochem. 1981;17(1):87-90. doi: 10.1002/jsscb.380170110.

Xeroderma pigmentosum: a human disease in which an initial stage of DNA repair is defective.着色性干皮病：一种人类疾病，其DNA修复的初始阶段存在缺陷。

Proc Natl Acad Sci U S A. 1969 Jun;63(2):428-35. doi: 10.1073/pnas.63.2.428.

Colony-forming ability of ultraviolet-irradiated xeroderma pigmentosum fibroblasts from different DNA repair complementation groups.来自不同DNA修复互补组的紫外线照射的着色性干皮病成纤维细胞的集落形成能力。

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Studies on a new case of xeroderma pigmentosum (XP3BR) from complementation group G with cellular sensitivity to ionizing radiation.对来自互补组G的一例新的着色性干皮病（XP3BR）患者的研究，该患者细胞对电离辐射敏感。

Carcinogenesis. 1980 Sep;1(9):745-51. doi: 10.1093/carcin/1.9.745.

引用本文的文献

The influence of DNA repair on neurological degeneration, cachexia, skin cancer and internal neoplasms: autopsy report of four xeroderma pigmentosum patients (XP-A, XP-C and XP-D).DNA 修复对神经退行性变、恶病质、皮肤癌和内脏肿瘤的影响：四名着色性干皮病患者（XP-A、XP-C 和 XP-D）的尸检报告。

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Deficient DNA repair capacity, a predisposing factor in breast cancer.DNA修复能力缺陷是乳腺癌的一个易感因素。

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Assessment of DNA damage and repair in specific genomic regions by quantitative immuno-coupled PCR.通过定量免疫偶联PCR评估特定基因组区域的DNA损伤与修复

Nucleic Acids Res. 1994 Jun 25;22(12):2351-9. doi: 10.1093/nar/22.12.2351.

Repair of radiation-induced DNA damage in nondividing populations of human diploid fibroblasts.人类二倍体成纤维细胞非分裂群体中辐射诱导的DNA损伤修复

Biophys J. 1980 Jun;30(3):399-413. doi: 10.1016/S0006-3495(80)85104-6.

XP patients from Germany: correlation of colony-forming ability, unscheduled DNA synthesis and single-strand breaks after UV damage in xeroderma pigmentosum fibroblasts.来自德国的着色性干皮病患者：着色性干皮病成纤维细胞经紫外线损伤后集落形成能力、非预定DNA合成与单链断裂的相关性

J Cancer Res Clin Oncol. 1982;104(3):263-86. doi: 10.1007/BF00406246.

Regulation of DNA repair in serum-stimulated xeroderma pigmentosum cells.血清刺激的着色性干皮病细胞中DNA修复的调控

J Cell Biol. 1984 Oct;99(4 Pt 1):1275-81. doi: 10.1083/jcb.99.4.1275.

Cytotoxic and mutagenic effects of specific carcinogen-DNA adducts in diploid human fibroblasts.特定致癌物-DNA加合物在二倍体人成纤维细胞中的细胞毒性和致突变作用。

Environ Health Perspect. 1985 Oct;62:145-55. doi: 10.1289/ehp.8562145.

Cell cycle-dependent strand bias for UV-induced mutations in the transcribed strand of excision repair-proficient human fibroblasts but not in repair-deficient cells.在切除修复功能正常的人成纤维细胞的转录链中，紫外线诱导的突变存在细胞周期依赖性链偏向性，但在修复缺陷细胞中则不存在。

Mol Cell Biol. 1991 Apr;11(4):1927-34. doi: 10.1128/mcb.11.4.1927-1934.1991.

Xeroderma pigmentosum neurological abnormalities correlate with colony-forming ability after ultraviolet radiation.着色性干皮病的神经学异常与紫外线照射后的集落形成能力相关。

Proc Natl Acad Sci U S A. 1978 Apr;75(4):1984-8. doi: 10.1073/pnas.75.4.1984.

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