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Mechanisms of inhibition of DNA replication by ultraviolet light in normal human and xeroderma pigmentosum fibroblasts.

作者信息

Kaufmann W K, Cleaver J E

出版信息

J Mol Biol. 1981 Jun 25;149(2):171-87. doi: 10.1016/0022-2836(81)90297-7.

DOI:10.1016/0022-2836(81)90297-7
PMID:7310880
Abstract
摘要

相似文献

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Mechanisms of inhibition of DNA replication by ultraviolet light in normal human and xeroderma pigmentosum fibroblasts.紫外线对正常人及着色性干皮病成纤维细胞DNA复制的抑制机制
J Mol Biol. 1981 Jun 25;149(2):171-87. doi: 10.1016/0022-2836(81)90297-7.
2
Typical xeroderma pigmentosum complementation group A fibroblasts have detectable ultraviolet light-induced unscheduled DNA synthesis.典型的着色性干皮病 A 互补组成纤维细胞具有可检测到的紫外线诱导的非程序性 DNA 合成。
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3
Xeroderma pigmentosum: a human disease in which an initial stage of DNA repair is defective.着色性干皮病：一种人类疾病，其DNA修复的初始阶段存在缺陷。
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Xeroderma pigmentosum: variants with normal DNA repair and normal sensitivity to ultraviolet light.着色性干皮病：具有正常DNA修复能力且对紫外线敏感性正常的变异型。
J Invest Dermatol. 1972 Mar;58(3):124-8. doi: 10.1111/1523-1747.ep12538913.
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DNA chain elongation and joining in normal human and xeroderma pigmentosum cells after ultraviolet irradiation.紫外线照射后正常人细胞和着色性干皮病细胞中的DNA链延伸与连接
Biophys J. 1972 Sep;12(9):1183-91. doi: 10.1016/S0006-3495(72)86154-X.
6
Different inherited levels of DNA repair replication in xeroderma pigmentosum cell strains after exposure to ultraviolet irradiation.着色性干皮病细胞株在紫外线照射后DNA修复复制的不同遗传水平。
Mutat Res. 1970 May;9(5):507-16. doi: 10.1016/0027-5107(70)90035-7.
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Colony-forming ability of ultraviolet-irradiated xeroderma pigmentosum fibroblasts from different DNA repair complementation groups.来自不同DNA修复互补组的紫外线照射的着色性干皮病成纤维细胞的集落形成能力。
Biochim Biophys Acta. 1976 Aug 18;442(2):147-53. doi: 10.1016/0005-2787(76)90485-8.
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Lethality and the induction and repair of DNA damage in far, mid or near UV-irradiated human fibroblasts: comparison of effects in normal, xeroderma pigmentosum and Bloom's syndrome cells.
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Steps in DNA chain elongation and joining after ultra-violet irradiation of human cells.紫外线照射人类细胞后DNA链延伸和连接的步骤。
Int J Radiat Biol Relat Stud Phys Chem Med. 1972 Nov;22(5):417-24. doi: 10.1080/09553007214551301.
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Xeroderma pigmentosum cells with normal levels of excision repair have a defect in DNA synthesis after UV-irradiation.具有正常切除修复水平的着色性干皮病细胞在紫外线照射后DNA合成存在缺陷。
Proc Natl Acad Sci U S A. 1975 Jan;72(1):219-23. doi: 10.1073/pnas.72.1.219.

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Cyclobutane Pyrimidine Dimer Density as a Predictive Biomarker of the Biological Effects of Ultraviolet Radiation in Normal Human Fibroblast.环丁烷嘧啶二聚体密度作为预测正常人类成纤维细胞中紫外线辐射生物学效应的生物标志物。
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