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Diabetic vascular dysfunctions: a model of excessive activation of protein kinase C.

作者信息

King G L, Ishii H, Koya D

机构信息

Research Division, Joslin Diabetes Center, Boston, Massachusetts, USA.

出版信息

Kidney Int Suppl. 1997 Sep;60:S77-85.

PMID:9285907
Abstract
摘要

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Diabetic vascular dysfunctions: a model of excessive activation of protein kinase C.
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Perturbed Biochemical Pathways and Associated Oxidative Stress Lead to Vascular Dysfunctions in Diabetic Retinopathy.受干扰的生化途径和相关的氧化应激导致糖尿病视网膜病变中的血管功能障碍。
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Therapeutic implications of curcumin in the prevention of diabetic retinopathy via modulation of anti-oxidant activity and genetic pathways.姜黄素通过调节抗氧化活性和遗传途径预防糖尿病视网膜病变的治疗意义。
Int J Physiol Pathophysiol Pharmacol. 2013 Dec 15;5(4):194-202.
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Myogenic tone and reactivity of cerebral arteries in type II diabetic BBZDR/Wor rat.
II型糖尿病BBZDR/Wor大鼠脑动脉的肌源性张力和反应性
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Human munc13 is a diacylglycerol receptor that induces apoptosis and may contribute to renal cell injury in hyperglycemia.人munc13是一种二酰基甘油受体，可诱导细胞凋亡，并可能导致高血糖症中的肾细胞损伤。
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