p53-mediated protective responses to UV irradiation.
作者信息
Smith M L, Fornace A J
机构信息
Laboratory of Molecular Pharmacology, Division of Basic Sciences, National Cancer Institute, National Institutes of Health, Building 37, Room 5D02, Bethesda, MD 20892, USA.
出版信息
Proc Natl Acad Sci U S A. 1997 Nov 11;94(23):12255-7. doi: 10.1073/pnas.94.23.12255.
Abstract
摘要
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本文引用的文献
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Enhancement of DNA repair in human skin cells by thymidine dinucleotides: evidence for a p53-mediated mammalian SOS response.胸腺嘧啶二核苷酸增强人类皮肤细胞中的DNA修复：p53介导的哺乳动物SOS反应的证据。
Proc Natl Acad Sci U S A. 1997 Nov 11;94(23):12627-32. doi: 10.1073/pnas.94.23.12627.
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Expression of wild-type p53 is required for efficient global genomic nucleotide excision repair in UV-irradiated human fibroblasts.野生型p53的表达是紫外线照射的人成纤维细胞中高效进行全基因组核苷酸切除修复所必需的。
J Biol Chem. 1997 Oct 31;272(44):28073-80. doi: 10.1074/jbc.272.44.28073.
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Conservation of the Chk1 checkpoint pathway in mammals: linkage of DNA damage to Cdk regulation through Cdc25.哺乳动物中Chk1检查点通路的保守性：通过Cdc25将DNA损伤与细胞周期蛋白依赖性激酶调控联系起来。
Science. 1997 Sep 5;277(5331):1497-501. doi: 10.1126/science.277.5331.1497.
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Suppression of UV-induced mutations by wild-type p53 protein in human osteosarcoma cells.野生型p53蛋白对人骨肉瘤细胞紫外线诱导突变的抑制作用。
Mutagenesis. 1997 May;12(3):191-4. doi: 10.1093/mutage/12.3.191.
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