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本文引用的文献
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VP16 targets an amino-terminal domain of HCF involved in cell cycle progression.
Mol Cell Biol. 1997 Oct;17(10):6139-46. doi: 10.1128/MCB.17.10.6139.
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Luman, a new member of the CREB/ATF family, binds to herpes simplex virus VP16-associated host cellular factor.
Mol Cell Biol. 1997 Sep;17(9):5117-26. doi: 10.1128/MCB.17.9.5117.
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Interdigitated residues within a small region of VP16 interact with Oct-1, HCF, and DNA.
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N-Oct 5 is generated by in vitro proteolysis of the neural POU-domain protein N-Oct 3.
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The CRE-binding protein dCREB-A is required for Drosophila embryonic development.
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Protein interactions in the herpes simplex virus type 1 VP16-induced complex: VP16 peptide inhibition and mutational analysis of host cell factor requirements.
J Virol. 1997 May;71(5):3886-94. doi: 10.1128/JVI.71.5.3886-3894.1997.
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A single-point mutation in HCF causes temperature-sensitive cell-cycle arrest and disrupts VP16 function.
Genes Dev. 1997 Mar 15;11(6):726-37. doi: 10.1101/gad.11.6.726.
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The Drosophila dCREB-A gene is required for dorsal/ventral patterning of the larval cuticle.
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Conformational alteration of Oct-1 upon DNA binding dictates selectivity in differential interactions with related transcriptional coactivators.
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Differential control of transcription by homologous homeodomain coregulators.
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