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脑梗死脑水肿手术治疗的试验——结合我们31例自身经验的综述(作者译)

[A trial of surgical management of brain edema in cerebral infarction--a review with our own experiences in 31 cases (author's transl)].

作者信息

Kakita K, Miyazaki T, Kadowaki H, Izawa M, Kubota S

出版信息

No Shinkei Geka. 1976 Mar;4(3):277-83.

PMID:944869
Abstract

We have analyzed the clinical course of 31 cases with cerebral infarction for 4 years. Of the 31 patients, 6 cases (19%) showed the brain edema on the cerebral angiograms as manifested by the shift of the midline arteries. The patients were divided into two groups, normotensive (15 cases) and hypertensive cerebral infarction (16 cases). The clinical and angiographic findings in the normotensive groups have been differed significantly from those of the hypertensive groups. The findings of brain edema on the angiograms were prominent in the hypertensive groups, and the clinical course was generally good and fair in the normotensive groups. We have reviewed the literatures about brain edema associated with cerebral infarction and have discussed on the mechanisms. From the study of our cases, it was concluded that systemic hypertension could be a facilitatory factor in the occurrence of brain edema in cerebral infarction. Furthermore, the management of massive cerebral infarction should be reduction of increased intracranial pressure and prevention of cerebral herniation. So, it will be necessary to perform a surgical treatment such as internal decompression; removal of the infarcted, necrotized area and excision of herniated tissue, and external decompression; removal of boneflap.

摘要

我们对31例脑梗死患者的临床病程进行了4年的分析。在这31例患者中,6例(19%)在脑血管造影上显示出脑水肿,表现为中线动脉移位。患者被分为两组,血压正常组(15例)和高血压性脑梗死组(16例)。血压正常组的临床和血管造影表现与高血压组有显著差异。血管造影上脑水肿的表现在高血压组更为突出,而血压正常组的临床病程总体较好。我们回顾了有关脑梗死相关脑水肿的文献并讨论了其机制。从我们的病例研究得出结论,系统性高血压可能是脑梗死中脑水肿发生的一个促进因素。此外,大面积脑梗死的治疗应是降低颅内压升高并预防脑疝形成。因此,有必要进行手术治疗,如内减压;切除梗死、坏死区域和突出组织,以及外减压;去除骨瓣。

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