Hijosa M, Esteban A, Sánchez Migallón M J, Grandas F
Servicio de Neurofisiología Clínica, Hospital Militar López Ulla, Madrid.
Neurologia. 1998 Jan;13(1):49-53.
A case that combines cerebral eyelid ptosis and blepharospasm secondary to cerebral hemisphere infarction is shown. EMG recording from the facial and eyelid muscles revealed lack of the levator palpebrae superioris, orbicularis oculi and corrugator muscles activity. Any voluntary opening attempt lead to a simultaneous contraction of the three muscles. Blink reflex responses were normal although showed facilitation features on the right side and mild inhibition characteristics from the left side. Median nerve SEP revealed a loss of precentral components (P22-N30) as well as a delay and amplitude decrease of N20. Transcranial magnetic stimulation disclosed a complete lesion of corticospinal pathway for right upper limb. In this case, a right hemisphere lesion caused an unusual eyelid motor abnormality: cerebral eyelid ptosis and blepharospasm induced by the voluntary eyelid opening.
本文展示了一例合并大脑半球梗死继发的脑性眼睑下垂和眼睑痉挛的病例。对面部和眼睑肌肉进行肌电图记录显示,提上睑肌、眼轮匝肌和皱眉肌缺乏活动。任何自主睁眼尝试都会导致这三块肌肉同时收缩。眨眼反射反应正常,尽管右侧表现为易化特征,左侧表现为轻度抑制特征。正中神经体感诱发电位显示中央前回成分(P22-N30)缺失,以及N20延迟和波幅降低。经颅磁刺激显示右侧上肢皮质脊髓通路完全受损。在该病例中,右侧半球病变导致了一种不寻常的眼睑运动异常:自主睁眼诱发的脑性眼睑下垂和眼睑痉挛。