介导T细胞活化诱导死亡的受体和配体。 - Suppr

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超能文献

Receptors and ligands that mediate activation-induced death of T cells.

作者信息

Alderson M R, Lynch D H

机构信息

Department of Immunology, Corixa Corporation, Seattle, WA 98104, USA.

出版信息

Springer Semin Immunopathol. 1998;19(3):289-300. doi: 10.1007/BF00787226.

DOI:10.1007/BF00787226

PMID:9540157

Abstract

摘要

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Receptors and ligands that mediate activation-induced death of T cells.介导T细胞活化诱导死亡的受体和配体。

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Differential regulation of the expression of CD95 ligand, receptor activator of nuclear factor-kappa B ligand (RANKL), TNF-related apoptosis-inducing ligand (TRAIL), and TNF-alpha during T cell activation.T细胞活化过程中CD95配体、核因子-κB受体激活剂配体（RANKL）、肿瘤坏死因子相关凋亡诱导配体（TRAIL）和肿瘤坏死因子-α表达的差异调节。

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ZAP-70 tyrosine kinase is required for the up-regulation of Fas ligand in activation-induced T cell apoptosis.ZAP-70酪氨酸激酶是激活诱导的T细胞凋亡中Fas配体上调所必需的。

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Zeta-associated protein of 70 kDa (ZAP-70), but not Syk, tyrosine kinase can mediate apoptosis of T cells through the Fas/Fas ligand, caspase-8 and caspase-3 pathways.70千道尔顿的ζ相关蛋白（ZAP-70）而非脾酪氨酸激酶（Syk），可通过Fas/Fas配体、半胱天冬酶-8和半胱天冬酶-3途径介导T细胞凋亡。

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Monocyte-driven activation-induced apoptotic cell death of human T-lymphotropic virus type I-infected T cells.单核细胞驱动的I型人类嗜T细胞病毒感染的T细胞激活诱导凋亡性细胞死亡

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Stimulation of phosphatidylinositol turnover is a key event for Fas-dependent, activation-induced apoptosis in human T lymphocytes.磷脂酰肌醇代谢的刺激是人类T淋巴细胞中Fas依赖性、激活诱导的细胞凋亡的关键事件。

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Fas ligand expression in islets of Langerhans does not confer immune privilege and instead targets them for rapid destruction.胰岛中Fas配体的表达不会赋予免疫特权，反而会将胰岛作为快速破坏的靶点。

Nat Med. 1997 Jul;3(7):738-43. doi: 10.1038/nm0797-738.

CD2 rescues T cells from T-cell receptor/CD3 apoptosis: a role for the Fas/Fas-L system.CD2可使T细胞免受T细胞受体/CD3介导的凋亡：Fas/Fas-L系统的作用。

Blood. 1997 May 15;89(10):3717-26.

Differential ability of T cell subsets to undergo activation-induced cell death.T细胞亚群经历活化诱导细胞死亡的差异能力。

Proc Natl Acad Sci U S A. 1997 May 27;94(11):5778-83. doi: 10.1073/pnas.94.11.5778.

The role of FasL-induced apoptosis in immune privilege.FasL诱导的细胞凋亡在免疫赦免中的作用。

Immunol Today. 1997 May;18(5):240-4. doi: 10.1016/s0167-5699(97)81663-5.

Unequal death in T helper cell (Th)1 and Th2 effectors: Th1, but not Th2, effectors undergo rapid Fas/FasL-mediated apoptosis.辅助性T细胞（Th）1和Th2效应细胞的死亡不均等：Th1效应细胞而非Th2效应细胞会经历快速的Fas/FasL介导的凋亡。

J Exp Med. 1997 May 19;185(10):1837-49. doi: 10.1084/jem.185.10.1837.

Role of Fas ligand (CD95L) in immune escape: the tumor cell strikes back.Fas配体（CD95L）在免疫逃逸中的作用：肿瘤细胞反击。

J Immunol. 1997 May 15;158(10):4521-4.

Transgenic expression of CD95 ligand on islet beta cells induces a granulocytic infiltration but does not confer immune privilege upon islet allografts.胰岛β细胞上CD95配体的转基因表达可诱导粒细胞浸润，但不能赋予胰岛同种异体移植物免疫特权。

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