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[桡肱骨髁上病理的神经生理学发现]

[Neurophysiologic findings in radio-humeral epicondyle pathology].

作者信息

Albrecht S, Cordis R, Kleihues H

机构信息

Abteilung für Orthopädie des Ev. Waldkrankenhaus Spandau.

出版信息

Sportverletz Sportschaden. 1998 Mar;12(1):8-14. doi: 10.1055/s-2007-993328.

Abstract

A number of authors assumed a compression syndrome of the radial nerve or its branches to be responsible for the heterogenous classified picture of radiohumeral epicondylopathy. Various structural and functional stenoses have been discussed as possible causes. We performed electromyographies on the extensor muscles, subdividing from the radial epicondyle and found significant (p < 0.05) changes in 27/51 patients regarding latency, velocity of neural conduction and rate of polyphasic potentials. Especially affected were the extensor carpi radialis brevis and extensor digitorum muscle. These findings were confirmed by torque measurements and histologic observations from both muscular and tendon biopsies. In summary, we believe the model of a neurogenous origin of radio-humeral epicondylopathy to be an appropriate explanation.

摘要

一些作者认为,桡神经或其分支的压迫综合征是导致桡肱上髁炎分类多样的原因。各种结构和功能狭窄已被讨论为可能的病因。我们对从桡侧上髁分出的伸肌进行了肌电图检查,发现51例患者中有27例在潜伏期、神经传导速度和多相电位发生率方面有显著(p < 0.05)变化。尤其受影响的是桡侧腕短伸肌和指伸肌。这些发现通过扭矩测量以及肌肉和肌腱活检的组织学观察得到了证实。总之,我们认为桡肱上髁炎的神经源性起源模型是一个恰当的解释。

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