Naeye R L
Am J Clin Pathol. 1976 Sep;66(3):526-30. doi: 10.1093/ajcp/66.3.526.
Fourteen of 28 sudden-infant-death syndrome (SIDS) victims had an abnormal proliferation of astroglial fibers in their brain stems and an enlarged mass of chromaffin cells in their adrenal medullas. Both of these abnormalities were associated with greater than normal muscle in the small pulmonary arteries, abnormal brown fat retention, and retained extramedullary erythropoiesis. These latter three findings are probably evidences of chronic alveolar hypoventilation and hypoxemia. The brain stem and adrenal abnormalities are probably also secondary to chronic hypoxemia.
28名婴儿猝死综合征（SIDS）受害者中有14名脑干中的星形胶质纤维异常增生，肾上腺髓质中的嗜铬细胞团增大。这两种异常均与小肺动脉中大于正常的肌肉、异常的棕色脂肪潴留以及髓外造血有关。后三项发现可能是慢性肺泡通气不足和低氧血症的证据。脑干和肾上腺异常可能也是慢性低氧血症的继发结果。
