Summers C G, Parker J C
Am J Forensic Med Pathol. 1981 Jun;2(2):121-7. doi: 10.1097/00000433-198106000-00006.
Abnormal central neural regulation of respiration may be involved in the pathogenesis of the sudden infant death syndrome (SIDS). A retrospective investigation of brain-stem morphology in 34 SIDS victims compared to well-matched controls revealed medullary gliosis in the reticular formation in 12% and a single microglial nodule suggestive of a viral infection in one infant. These findings support the need for more extensive prospective neuropathological investigations in SIDS utilizing more sophisticated techniques.
呼吸的中枢神经调节异常可能与婴儿猝死综合征（SIDS）的发病机制有关。一项对34例SIDS受害者脑干形态的回顾性调查，并与匹配良好的对照组进行比较，结果显示，12%的受害者网状结构存在延髓胶质增生，一名婴儿有单个小胶质结节，提示病毒感染。这些发现支持了利用更先进技术对SIDS进行更广泛前瞻性神经病理学研究的必要性。
