暴发性肝衰竭时钠排泄异常及其他肾功能障碍
Abnormalities of sodium excretion and other disorders of renal function in fulminant hepatic failure.
作者信息
Wilkinson S P, Arroyo V A, Moodie H, Blendis L M, Williams R
出版信息
Gut. 1976 Jul;17(7):501-5. doi: 10.1136/gut.17.7.501.
Abstract
Renal function was evaluated in 40 patients with fulminant hepatic failure, They were divided into two groups on the basis of glomerular filtration rates greater than 40 ml/min or less than 25 ml/min. A number of patients in group 1 had markedly abnormal renal retention of sodium together with a reduced free water clearance and low potassium excretion which could be explained by increased proximal tubular reabsorption of sodium. The patients in group 2 had evidence that renal tubular integrity was maintained when the glomerular filtration rate was greater than or equal ml/min (functional renal failure), but evidence of tubular damage was present when this was less than 3 ml/min (acute tubular necrosis).
摘要
对40例暴发性肝衰竭患者的肾功能进行了评估。根据肾小球滤过率大于40ml/分钟或小于25ml/分钟将他们分为两组。第1组中的一些患者存在明显异常的肾钠潴留，同时自由水清除率降低和钾排泄减少，这可以用近端肾小管对钠的重吸收增加来解释。第2组患者在肾小球滤过率大于或等于ml/分钟时（功能性肾衰竭）有肾小管完整性维持的证据，但当肾小球滤过率小于3ml/分钟时（急性肾小管坏死）则有肾小管损伤的证据。 （注：原文中“greater than or equal ml/min”表述有误，可能遗漏了具体数值）
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